Dysfunctional synapse in Alzheimer's disease – A focus on NMDA receptorsSandra I. Mota, A. Cristina Rego|Neuropharmacology|2013Cited by 201
Insulin and IGF-1 improve mitochondrial function in a PI-3K/Akt-dependent manner and reduce mitochondrial generation of reactive oxygen species in Huntington’s disease knock-in striatal cellsMárcio Ribeiro, A. Cristina Rego|Free Radical Biology and Medicine|2014Cited by 138
The R6 lines of transgenic mice: A model for screening new therapies for Huntington's diseaseJoana Gil‐Mohapel, A. Cristina Rego|Brain Research Reviews|2008Cited by 63
Histone Deacetylase Inhibitors Protect Against Pyruvate Dehydrogenase Dysfunction in Huntington's DiseaseLuana Naia, A. Cristina Rego|Journal of Neuroscience|2017Cited by 62
Uncovering the Early Events Associated with Oligomeric Aβ-Induced Src ActivationSandra I. Mota, A. Cristina Rego|Antioxidants|2023Cited by 4