Brain Insulin Signaling Is Increased in Insulin-Resistant States and Decreases in FOXOs and PGC-1α and Increases in Aβ1–40/42 and Phospho-Tau May Abet Alzheimer DevelopmentMini P. Sajan, Robert V. Farese, Shijie Song et al.|Diabetes|2016Cited by 87
Hepatic insulin resistance in ob/ob mice involves increases in ceramide, aPKC activity, and selective impairment of Akt-dependent FoxO1 phosphorylationMini P. Sajan, Robert V. Farese, Mackenzie C. Lee et al.|Journal of Lipid Research|2014Cited by 61
Akt-Dependent Phosphorylation of Hepatic FoxO1 Is Compartmentalized on a WD40/ProF Scaffold and Is Selectively Inhibited by aPKC in Early Phases of Diet-Induced ObesityMini P. Sajan, Robert V. Farese, Mildred Acevedo‐Duncan et al.|Diabetes|2014Cited by 34
Requirements for Pseudosubstrate Arginine Residues during Autoinhibition and Phosphatidylinositol 3,4,5-(PO4)3-dependent Activation of Atypical PKCRobert A. Ivey, Robert V. Farese, Mini P. Sajan|Journal of Biological Chemistry|2014Cited by 29
BMI-related progression of atypical PKC-dependent aberrations in insulin signaling through IRS-1, Akt, FoxO1 and PGC-1α in livers of obese and type 2 diabetic humansMini P. Sajan, Robert V. Farese, Robert A. Ivey|Metabolism|2015Cited by 23