MYC pathway activation in triple-negative breast cancer is synthetic lethal with CDK inhibitionDai Horiuchi, Andrei Goga|The Journal of Experimental Medicine|2012Cited by 395
Switching Cdk2 On or Off with Small Molecules to Reveal Requirements in Human Cell ProliferationKarl A. Merrick, Robert P. Fisher, Lara Wohlbold et al.|Molecular Cell|2011Cited by 85
Chemical-genetic analysis of cyclin dependent kinase 2 function reveals an important role in cellular transformation by multiple oncogenic pathwaysDai Horiuchi, Andrei Goga, Chao Zhang et al.|Proceedings of the National Academy of Sciences|2012Cited by 75
Linking Tumor Mutations to Drug Responses via a Quantitative Chemical–Genetic Interaction MapMaria M. Martins, Sourav Bandyopadhyay, Alicia Y. Zhou et al.|Cancer Discovery|2014Cited by 62
S100A8/A9 predicts response to PIM kinase and PD-1/PD-L1 inhibition in triple-negative breast cancer mouse modelsLauren R. Begg, Dai Horiuchi|Communications Medicine|2024Cited by 7