Full Reversal of Alzheimer's Disease-Like Phenotype in a Mouse Model with Conditional Overexpression of Glycogen Synthase Kinase-3Tobías Engel, José J. Lucas|Journal of Neuroscience|2006Cited by 253
Ubiquitin–proteasome system involvement in Huntington’s diseaseZaira Ortega, José J. Lucas|Frontiers in Molecular Neuroscience|2014Cited by 107
Proteasomal Expression, Induction of Immunoproteasome Subunits, and Local MHC Class I Presentation in Myofibrillar Myopathy and Inclusion Body MyositisIsidró Ferrer, Montse Olivé, Dolores Moreno et al.|Journal of Neuropathology & Experimental Neurology|2004Cited by 97
Acute Polyglutamine Expression in Inducible Mouse Model Unravels Ubiquitin/Proteasome System Impairment and Permanent Recovery Attributable to Aggregate FormationZaira Ortega, José J. Lucas|Journal of Neuroscience|2010Cited by 94
NFAT/Fas signaling mediates the neuronal apoptosis and motor side effects of GSK-3 inhibition in a mouse model of lithium therapyRaquel Gómez‐Sintes, José J. Lucas|Journal of Clinical Investigation|2010Cited by 81