Insulin Sensitivity and Mitochondrial Function Are Improved in Children With Burn Injury During a Randomized Controlled Trial of FenofibrateMelanie Cree‐Green, Robert R. Wolfe, Jennifer J. Zwetsloot et al.|Annals of Surgery|2007Cited by 112
Contraction of insulin-resistant muscle normalizes insulin action in association with increased mitochondrial activity and fatty acid catabolismJohn P. Thyfault, G. Lynis Dohm, Jennifer J. Zwetsloot et al.|American Journal of Physiology-Cell Physiology|2006Cited by 88
PPAR-α agonism improves whole body and muscle mitochondrial fat oxidation, but does not alter intracellular fat concentrations in burn trauma children in a randomized controlled trialMelanie Cree‐Green, Robert R. Wolfe, Bradley R. Newcomer et al.|Nutrition & Metabolism|2007Cited by 52
Insulin Sensitivity is Related to Fat Oxidation and Protein Kinase C Activity in Children With Acute Burn InjuryMelanie Cree‐Green, Robert R. Wolfe, Jennifer J. Zwetsloot et al.|Journal of Burn Care & Research|2008Cited by 14
PPAR‐α agonism improves whole body and muscle mitochondrial fat oxidation, but does not alter intracellular fat concentrations in burn trauma childrenMelanie Cree‐Green, Robert R. Wolfe, Bradley R. Newcomer et al.|The FASEB Journal|2007Cited by 1