Claudin-16 and claudin-19 interact and form a cation-selective tight junction complexJianghui Hou, Daniel A. Goodenough, Aparna Renigunta et al.|Journal of Clinical Investigation|2008Cited by 333
Defects in KCNJ16 Cause a Novel Tubulopathy with Hypokalemia, Salt Wasting, Disturbed Acid-Base Homeostasis, and Sensorineural DeafnessKarl P. Schlingmann, Martin Konrad, Sinthura Mahendran et al.|Journal of the American Society of Nephrology|2021Cited by 72
Type, location and zygosity of <i>KCNJ16</i> mutations may determine the clinical severity of Hypokalemic Tubulopathy and Deafness (HkTD)Leire Gondra, Aparna Renigunta|medRxiv|2026Cited by 0