Overexpression of wild type or a Q311E mutant <i>MB21D2</i> promotes a pro‐oncogenic phenotype in HNSCCDaniel E. Gracilla, Jim Jinn‐Chyuan Sheu|Molecular Oncology|2020Cited by 11
FET Fusion Oncoproteins Disrupt Physiologic DNA Repair and Create a Targetable Opportunity for ATR Inhibitor TherapyDaniel E. Gracilla, Asmin Tulpule|Cancer Research|2026Cited by 0