Molecular and cellular signatures differentiate Parkinson’s disease from Parkinson’s disease with dementiaAine Fairbrother-Browne, Mina Ryten, Melissa Grant‐Peters et al.|bioRxiv (Cold Spring Harbor Laboratory)|2025Cited by 4
Transcriptomic analysis of repeat expansion-ataxias uncovers distinct non-neuronal cell type-specific signatures of disease across the human brainZhongbo Chen, Mina Ryten, Amy R. Hicks et al.|bioRxiv (Cold Spring Harbor Laboratory)|2025Cited by 2
TDP-43 loss of function drives aberrant splicing in Parkinson’s diseaseJonathan Brenton, Mina Ryten, Jordan Follett et al.|bioRxiv (Cold Spring Harbor Laboratory)|2025Cited by 1
17q21.31 locus regulates Parkinson’s disease relevant pathways through <i>KANSL1</i> activityAmy R. Hicks, Mina Ryten, Benjamin O’Callaghan et al.|bioRxiv (Cold Spring Harbor Laboratory)|2025Cited by 0
CANVAS causing AAGGG repeat expansions cause tissue-specific reduction in RFC1 expression and increase sensitivity to DNA damageRiccardo Curró, Andrea Cortese, Glenda Paola Grupelli et al.|bioRxiv (Cold Spring Harbor Laboratory)|2025Cited by 0