Cell Signaling Technology

Dopamine beta-Hydroxylase (DBH) Antibody

#8586 · $317

100
Citing papers
8.3k
Combined citations
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Research areas

Autophagy in Disease and Therapy33Cancer-related Molecular Pathways10DNA Repair Mechanisms9Epigenetics and DNA Methylation7Ubiquitin and proteasome pathways7Cell death mechanisms and regulation6Cancer-related gene regulation5Cell Adhesion Molecules Research5

Papers citing this product

1
ER stress (PERK/eIF2α phosphorylation) mediates the polyglutamine-induced LC3 conversion, an essential step for autophagy formation
Cell Death and Differentiation·2006·923 cit.·DOI
2
Beclin 1 Gene Transfer Activates Autophagy and Ameliorates the Neurodegenerative Pathology in α-Synuclein Models of Parkinson's and Lewy Body Diseases
Journal of Neuroscience·2009·581 cit.·DOI
3
A Non-canonical MEK/ERK Signaling Pathway Regulates Autophagy via Regulating Beclin 1
Journal of Biological Chemistry·2009·321 cit.·DOI
4
Autophagy promotes T-cell survival through degradation of proteins of the cell death machinery
Cell Death and Differentiation·2011·213 cit.·DOI
5
Reelin Stabilizes the Actin Cytoskeleton of Neuronal Processes by Inducing n-Cofilin Phosphorylation at Serine3
Journal of Neuroscience·2009·203 cit.·DOI
6
TREM2hi resident macrophages protect the septic heart by maintaining cardiomyocyte homeostasis
Nature Metabolism·2023·188 cit.·DOI
7
A NOTCH feed-forward loop drives reprogramming from adrenergic to mesenchymal state in neuroblastoma
Nature Communications·2019·171 cit.·DOI
8
Apolipoprotein E-Containing Lipoproteins Protect Neurons from Apoptosis via a Signaling Pathway Involving Low-Density Lipoprotein Receptor-Related Protein-1
Journal of Neuroscience·2007·170 cit.·DOI
9
Microcephalin Is a DNA Damage Response Protein Involved in Regulation of CHK1 and BRCA1
Journal of Biological Chemistry·2004·170 cit.·DOI
10
Mitochondrial Translocation of p53 Mediates Release of Cytochrome <i>c</i> and Hippocampal CA1 Neuronal Death after Transient Global Cerebral Ischemia in Rats
Journal of Neuroscience·2006·161 cit.·DOI