Cell Signaling Technology

Phospho-alpha-Synuclein (Ser129) (D1R1R) Rabbit Monoclonal Antibody

#23706 · $193

44
Citing papers
696
Combined citations
Supplier page Find this product

Research areas

Parkinson's Disease Mechanisms and Treatments21Alzheimer's disease research and treatments9Cancer-related molecular mechanisms research8Neurological disorders and treatments6Nuclear Receptors and Signaling5Autophagy in Disease and Therapy5Nerve injury and regeneration4Cellular transport and secretion4

Papers citing this product

1
Cofilin 1 promotes the pathogenicity and transmission of pathological α-synuclein in mouse models of Parkinson’s disease
npj Parkinson s Disease·2022·77 cit.·DOI
2
Phosphorylation of phase‐separated p62 bodies by <scp>ULK1</scp> activates a redox‐independent stress response
The EMBO Journal·2023·70 cit.·DOI
3
&lt;p&gt;Phosphorylation of Tau and α-Synuclein Induced Neurodegeneration in MPTP Mouse Model of Parkinson’s Disease&lt;/p&gt;
Neuropsychiatric Disease and Treatment·2020·59 cit.·DOI
4
Long noncoding RNA Malat1 protects against osteoporosis and bone metastasis
Nature Communications·2024·58 cit.·DOI
5
Rapamycin reverses ferroptosis by increasing autophagy in MPTP/MPP+-induced models of Parkinson’s disease
Neural Regeneration Research·2023·46 cit.·DOI
6
Sex-Dependent Effects of Intestinal Microbiome Manipulation in a Mouse Model of Alzheimer’s Disease
Cells·2021·36 cit.·DOI
7
Polo-like kinase 2 inhibition reduces serine-129 phosphorylation of physiological nuclear alpha-synuclein but not of the aggregated alpha-synuclein
PLoS ONE·2021·35 cit.·DOI
8
Clemastine Promotes Differentiation of Oligodendrocyte Progenitor Cells Through the Activation of ERK1/2 via Muscarinic Receptors After Spinal Cord Injury
Frontiers in Pharmacology·2022·27 cit.·DOI
9
MED13 and glycolysis are conserved modifiers of α-synuclein-associated neurodegeneration
Cell Reports·2022·23 cit.·DOI
10
Promotion of TLR7-MyD88-dependent inflammation and autoimmunity in mice through stem-loop changes in Lnc-Atg16l1
Nature Communications·2024·23 cit.·DOI

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