Cell Signaling Technology

RalA Antibody

#3526 · $317

65
Evidence Score · Well-Established
Used by 192 independent labs across 200 papers. How we score this →
100
Citing papers
10.3k
Combined citations
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Research areas

MicroRNA in disease regulation15Circular RNAs in diseases9Cancer-related molecular mechanisms research8RNA modifications and cancer7Circadian rhythm and melatonin6Immune cells in cancer5Photoreceptor and optogenetics research5RNA Research and Splicing5

Papers citing this product

1
Myeloid-derived suppressor cell development is regulated by a STAT/IRF-8 axis
Journal of Clinical Investigation·2013·319 cit.·DOI
2
Targeted inhibition of p38 MAPK promotes hypertrophic cardiomyopathy through upregulation of calcineurin-NFAT signaling
Journal of Clinical Investigation·2003·306 cit.·DOI
3
Extracellular Vesicles Derived from Bone Marrow Mesenchymal Stem Cells Protect against Experimental Colitis via Attenuating Colon Inflammation, Oxidative Stress and Apoptosis
PLoS ONE·2015·280 cit.·DOI
4
Deregulated MYC expression induces dependence upon AMPK-related kinase 5
Nature·2012·278 cit.·DOI
5
<i>Porphyromonas gingivalis</i>promotes invasion of oral squamous cell carcinoma through induction of proMMP9 and its activation
Cellular Microbiology·2013·250 cit.·DOI
6
MicroRNA-99a Inhibits Hepatocellular Carcinoma Growth and Correlates with Prognosis of Patients with Hepatocellular Carcinoma
Journal of Biological Chemistry·2011·248 cit.·DOI
7
IL-1 receptor antagonist ameliorates inflammasome-dependent inflammation in murine and human cystic fibrosis
Nature Communications·2016·232 cit.·DOI
8
β-Caryophyllene Inhibits Dextran Sulfate Sodium-Induced Colitis in Mice through CB2 Receptor Activation and PPARγ Pathway
American Journal Of Pathology·2011·228 cit.·DOI
9
A Novel Signaling Network Essential for Regulating Pseudomonas aeruginosa Biofilm Development
PLoS Pathogens·2009·213 cit.·DOI
10
The role of the Grb2–p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis
Journal of Clinical Investigation·2003·212 cit.·DOI

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