Cell Signaling Technology

SMARCA1 (D4Q7V) Rabbit Monoclonal Antibody

#12483 · $161

100
Citing papers
3.3k
Combined citations
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Research areas

Cancer-related molecular mechanisms research11Autophagy in Disease and Therapy8NF-κB Signaling Pathways7Inflammatory mediators and NSAID effects6Cancer-related Molecular Pathways5Circular RNAs in diseases5Alzheimer's disease research and treatments5Epigenetics and DNA Methylation5

Papers citing this product

1
Resveratrol inhibits phorbol ester-induced expression of COX-2 and activation of NF-κB in mouse skin by blocking IκB kinase activity
Carcinogenesis·2006·266 cit.·DOI
2
Identification of longitudinally dynamic biomarkers in Alzheimer’s disease cerebrospinal fluid by targeted proteomics
Molecular Neurodegeneration·2014·150 cit.·DOI
3
Impaired neutrophil activity and increased susceptibility to bacterial infection in mice lacking glucose-6-phosphatase–β
Journal of Clinical Investigation·2007·121 cit.·DOI
4
The mitochondrial-derived peptide humanin activates the ERK1/2, AKT, and STAT3 signaling pathways and has age-dependent signaling differences in the hippocampus
Oncotarget·2016·114 cit.·DOI
5
Expansion of the ISWI chromatin remodeler family with new active complexes
EMBO Reports·2017·105 cit.·DOI
6
IRE1 signaling increases PERK expression during chronic ER stress
Cell Death and Disease·2024·80 cit.·DOI
7
Extracellular Vesicles Derived from Kefir Grain Lactobacillus Ameliorate Intestinal Inflammation via Regulation of Proinflammatory Pathway and Tight Junction Integrity
Biomedicines·2020·78 cit.·DOI
8
Humulone inhibits phorbol ester-induced COX-2 expression in mouse skin by blocking activation of NF-κB and AP-1: IκB kinase and c-Jun-N-terminal kinase as respective potential upstream targets
Carcinogenesis·2007·78 cit.·DOI
9
Ursolic and Oleanolic Acids Induce Mitophagy in A549 Human Lung Cancer Cells
Molecules·2019·74 cit.·DOI
10
MG53 suppresses tumor progression and stress granule formation by modulating G3BP2 activity in non-small cell lung cancer
Molecular Cancer·2021·70 cit.·DOI

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