Cell Signaling Technology

Cofilin Activation Antibody Sampler Kit

#8354 · $650

100
Citing papers
9.1k
Combined citations
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Research areas

Neuroinflammation and Neurodegeneration Mechanisms40Alzheimer's disease research and treatments30Neurogenesis and neuroplasticity mechanisms19Nerve injury and regeneration8RNA Research and Splicing7Pluripotent Stem Cells Research6Immune Response and Inflammation5Immune cells in cancer5

Papers citing this product

1
Regulation of exosome secretion by Rab35 and its GTPase-activating proteins TBC1D10A–C
The Journal of Cell Biology·2010·812 cit.·DOI
2
Adult-Born Hippocampal Dentate Granule Cells Undergoing Maturation Modulate Learning and Memory in the Brain
Journal of Neuroscience·2009·492 cit.·DOI
3
Functional Impairment of Microglia Coincides with Beta-Amyloid Deposition in Mice with Alzheimer-Like Pathology
PLoS ONE·2013·477 cit.·DOI
4
Sustained Interleukin-1β Overexpression Exacerbates Tau Pathology Despite Reduced Amyloid Burden in an Alzheimer's Mouse Model
Journal of Neuroscience·2013·374 cit.·DOI
5
An Effector-Reduced Anti- -Amyloid (A ) Antibody with Unique A  Binding Properties Promotes Neuroprotection and Glial Engulfment of A 
Journal of Neuroscience·2012·309 cit.·DOI
6
Microglial p38α MAPK is a key regulator of proinflammatory cytokine up-regulation induced by toll-like receptor (TLR) ligands or beta-amyloid (Aβ)
Journal of Neuroinflammation·2011·283 cit.·DOI
7
SIRT1 Deficiency in Microglia Contributes to Cognitive Decline in Aging and Neurodegeneration via Epigenetic Regulation of IL-1β
Journal of Neuroscience·2015·273 cit.·DOI
8
Adiponectin suppresses amyloid-β oligomer (AβO)-induced inflammatory response of microglia via AdipoR1-AMPK-NF-κB signaling pathway
Journal of Neuroinflammation·2019·191 cit.·DOI
9
A Critical Role of Autophagy in Regulating Microglia Polarization in Neurodegeneration
Frontiers in Aging Neuroscience·2018·179 cit.·DOI
10
Telomere Shortening in Neural Stem Cells Disrupts Neuronal Differentiation and Neuritogenesis
Journal of Neuroscience·2009·178 cit.·DOI