Cell Signaling Technology

CNOT3 (D4K4N) Rabbit Monoclonal Antibody

#40608 · $161

26
Citing papers
1.2k
Combined citations
Supplier page Find this product

Research areas

Cell death mechanisms and regulation18NF-κB Signaling Pathways10Immune Response and Inflammation3Ubiquitin and proteasome pathways2Inflammasome and immune disorders2Autophagy in Disease and Therapy2interferon and immune responses2Cancer Research and Treatments1

Papers citing this product

1
c-IAP1 and c-IAP2 Are Critical Mediators of Tumor Necrosis Factor α (TNFα)-induced NF-κB Activation
Journal of Biological Chemistry·2008·536 cit.·DOI
2
Caspase-8 prevents sustained activation of NF-κB in monocytes undergoing macrophagic differentiation
Blood·2006·125 cit.·DOI
3
MCL-1 as a Buffer for Proapoptotic BCL-2 Family Members during TRAIL-induced Apoptosis
Journal of Biological Chemistry·2007·108 cit.·DOI
4
Kinome profiling of non-canonical TRAIL signaling reveals RIP1-Src-STAT3 dependent invasion in resistant non-small cell lung cancer cells
Journal of Cell Science·2012·70 cit.·DOI
5
Dominant Negative Effects of Tumor Necrosis Factor (TNF)-related Apoptosis-inducing Ligand (TRAIL) Receptor 4 on TRAIL Receptor 1 Signaling by Formation of Heteromeric Complexes
Journal of Biological Chemistry·2014·52 cit.·DOI
6
Suppression of death receptor 5 enhances cancer cell invasion and metastasis through activation of caspase-8/TRAF2-mediated signaling
Oncotarget·2015·49 cit.·DOI
7
TRAIL-activated stress kinases suppress apoptosis through transcriptional upregulation of MCL-1
Cell Death and Differentiation·2010·43 cit.·DOI
8
Differential activation of JNK1 isoforms by TRAIL receptors modulate apoptosis of colon cancer cell lines
British Journal of Cancer·2009·41 cit.·DOI
9
Proteasome Inhibition Can Impair Caspase-8 Activation upon Submaximal Stimulation of Apoptotic Tumor Necrosis Factor-related Apoptosis Inducing Ligand (TRAIL) Signaling
Journal of Biological Chemistry·2012·40 cit.·DOI
10
TAK1 Is Required for Survival of Mouse Fibroblasts Treated with TRAIL, and Does So by NF-κB Dependent Induction of cFLIPL
PLoS ONE·2010·25 cit.·DOI

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