Cell Signaling Technology

Cyclin E2 Antibody

#4132 · western blot · $136,317

100
Citing papers
12.2k
Combined citations
Supplier page Find this product

Research areas

HER2/EGFR in Cancer Research16PI3K/AKT/mTOR signaling in cancer10MicroRNA in disease regulation9Neuroinflammation and Neurodegeneration Mechanisms8Cell death mechanisms and regulation8Monoclonal and Polyclonal Antibodies Research8Cancer-related molecular mechanisms research7Epigenetics and DNA Methylation6

Papers citing this product

1
Acquired CDK6 amplification promotes breast cancer resistance to CDK4/6 inhibitors and loss of ER signaling and dependence
Oncogene·2016·430 cit.·DOI
2
Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression
Scientific Reports·2015·356 cit.·DOI
3
TREM2 deficiency exacerbates tau pathology through dysregulated kinase signaling in a mouse model of tauopathy
Molecular Neurodegeneration·2017·332 cit.·DOI
4
Overlapping cleavage motif selectivity of caspases: implications for analysis of apoptotic pathways
Cell Death and Differentiation·2007·310 cit.·DOI
5
Amino Acid-Induced Translation of TOP mRNAs Is Fully Dependent on Phosphatidylinositol 3-Kinase-Mediated Signaling, Is Partially Inhibited by Rapamycin, and Is Independent of S6K1 and rpS6 Phosphorylation
Molecular and Cellular Biology·2001·298 cit.·DOI
6
Cardiolipin provides an essential activating platform for caspase-8 on mitochondria
The Journal of Cell Biology·2008·288 cit.·DOI
7
PTEN, PIK3CA, p-AKT, and p-p70S6K Status
American Journal Of Pathology·2010·285 cit.·DOI
8
Characterization of a novel cell line established from a patient with Herceptin-resistant breast cancer
Molecular Cancer Therapeutics·2004·269 cit.·DOI
9
The miR-106b-25 Polycistron, Activated by Genomic Amplification, Functions as an Oncogene by Suppressing p21 and Bim
Gastroenterology·2009·265 cit.·DOI
10
The antidiabetic drug metformin suppresses HER2 (erbB-2) oncoprotein overexpression via inhibition of the mTOR effector p70S6K1 in human breast carcinoma cells
Cell Cycle·2009·251 cit.·DOI

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