Cell Signaling Technology

MHC Class II (I-A/I-E) (M5/114.15.2) Rat Monoclonal Antibody (FITC Conjugate)

#42594 · $151

13
Citing papers
631
Combined citations
Supplier page Find this product

Research areas

Dermatology and Skin Diseases7Psoriasis: Treatment and Pathogenesis7Urticaria and Related Conditions2Cytokine Signaling Pathways and Interactions2T-cell and B-cell Immunology2Toxin Mechanisms and Immunotoxins2Ferroptosis and cancer prognosis1Cancer Immunotherapy and Biomarkers1

Papers citing this product

1
Increased Expression of Interleukin-36, a Member of the Interleukin-1 Cytokine Family, in Inflammatory Bowel Disease
Inflammatory Bowel Diseases·2016·129 cit.·DOI
2
IL-38 has an anti-inflammatory action in psoriasis and its expression correlates with disease severity and therapeutic response to anti-IL-17A treatment
Cell Death and Disease·2018·128 cit.·DOI
3
IL-36γ signaling controls the induced regulatory T cell–Th9 cell balance via NFκB activation and STAT transcription factors
Mucosal Immunology·2017·93 cit.·DOI
4
<scp>IL</scp>‐36γ has proinflammatory effects on human endothelial cells
Experimental Dermatology·2016·77 cit.·DOI
5
IL-36γ drives skin toxicity induced by EGFR/MEK inhibition and commensal Cutibacterium acnes
Journal of Clinical Investigation·2019·57 cit.·DOI
6
Interleukin-36γ is expressed by neutrophils and can activate microglia, but has no role in experimental autoimmune encephalomyelitis
Journal of Neuroinflammation·2015·47 cit.·DOI
7
Structural insights into heme binding to IL-36α proinflammatory cytokine
Scientific Reports·2019·34 cit.·DOI
8
Interleukin (IL)-1β Is a Strong Inducer of IL-36γ Expression in Human Colonic Myofibroblasts
PLoS ONE·2015·30 cit.·DOI
9
Designed DNA-Encoded IL-36 Gamma Acts as a Potent Molecular Adjuvant Enhancing Zika Synthetic DNA Vaccine-Induced Immunity and Protection in a Lethal Challenge Model
Vaccines·2019·19 cit.·DOI
10
Production of biologically active IL‐36 family cytokines through insertion of N‐terminal caspase cleavage motifs
FEBS Open Bio·2016·16 cit.·DOI

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