Cell Signaling Technology

Phospho-PKD/PKC mu (Ser744/748) Antibody

#2054 · $152,873

62

Evidence Score · Well-Established

Used by 98 independent labs across 102 papers. How we score this →

100

Citing papers

21.9k

Combined citations

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Research areas

Hippo pathway signaling and YAP/TAZ15 Cancer-related molecular mechanisms research11 RNA modifications and cancer8 Wnt/β-catenin signaling in development and cancer8 Epigenetics and DNA Methylation8 Cancer-related gene regulation7 Ubiquitin and proteasome pathways7 MicroRNA in disease regulation6

Papers citing this product

1

<i>miR-146a</i> is a significant brake on autoimmunity, myeloproliferation, and cancer in mice

The Journal of Experimental Medicine·2011·865 cit.·DOI

2

The Hippo pathway target, YAP, promotes metastasis through its TEAD-interaction domain

Proceedings of the National Academy of Sciences·2012·683 cit.·DOI

3

Copper-dependent autophagic degradation of GPX4 drives ferroptosis

Autophagy·2023·582 cit.·DOI

4

miR-124 and miR-203 are epigenetically silenced tumor-suppressive microRNAs in hepatocellular carcinoma

Carcinogenesis·2009·575 cit.·DOI

5

Inhibition of “self” engulfment through deactivation of myosin-II at the phagocytic synapse between human cells

The Journal of Cell Biology·2008·481 cit.·DOI

6

NF-κB Activation Represses Tumor Necrosis Factor-α-induced Autophagy

Journal of Biological Chemistry·2006·462 cit.·DOI

7

Mechanistic and prognostic significance of aberrant methylation in the molecular pathogenesis of human hepatocellular carcinoma

Journal of Clinical Investigation·2007·395 cit.·DOI

8

Epigenetic Changes Induced by Reactive Oxygen Species in Hepatocellular Carcinoma: Methylation of the E-cadherin Promoter

Gastroenterology·2008·382 cit.·DOI

9

Astrocytic Dysfunction in Epileptogenesis: Consequence of Altered Potassium and Glutamate Homeostasis?

Journal of Neuroscience·2009·343 cit.·DOI

10

Central role of RAGE-dependent neointimal expansion in arterial restenosis

Journal of Clinical Investigation·2003·342 cit.·DOI

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