Cell Signaling Technology

HS1 (D83A8) Rabbit Monoclonal Antibody

#3890 · western blot · $176,384

65
Evidence Score · Well-Established
Used by 486 independent labs across 521 papers. How we score this →
100
Citing papers
14.1k
Combined citations
Supplier page Find this product

Research areas

PI3K/AKT/mTOR signaling in cancer10Cancer-related molecular mechanisms research8Immune Cell Function and Interaction6MicroRNA in disease regulation5Mitochondrial Function and Pathology5Immunotherapy and Immune Responses4Melanoma and MAPK Pathways4Cancer, Hypoxia, and Metabolism4

Papers citing this product

1
Resveratrol Reduces the Proinflammatory Effects and Lipopolysaccharide- Induced Expression of HMGB1 and TLR4 in RAW264.7 Cells
Cellular Physiology and Biochemistry·2014·3.3k cit.·DOI
2
Pten Dose Dictates Cancer Progression in the Prostate
PLoS Biology·2003·667 cit.·DOI
3
PKM2 regulates the Warburg effect and promotes HMGB1 release in sepsis
Nature Communications·2014·511 cit.·DOI
4
Targeting AKT/mTOR and ERK MAPK signaling inhibits hormone-refractory prostate cancer in a preclinical mouse model
Journal of Clinical Investigation·2008·387 cit.·DOI
5
JAK/STAT1 signaling promotes HMGB1 hyperacetylation and nuclear translocation
Proceedings of the National Academy of Sciences·2014·362 cit.·DOI
6
Nucleocytoplasmic Shuttling of HMGB1 Is Regulated by Phosphorylation That Redirects It toward Secretion
The Journal of Immunology·2006·362 cit.·DOI
7
Human Umbilical Cord Mesenchymal Stem Cells Reduce Fibrosis of Bleomycin-Induced Lung Injury
American Journal Of Pathology·2009·361 cit.·DOI
8
Circulating and intrahepatic antiviral B cells are defective in hepatitis B
Journal of Clinical Investigation·2018·322 cit.·DOI
9
A computational model on the modulation of mitogen-activated protein kinase (MAPK) and Akt pathways in heregulin-induced ErbB signalling
Biochemical Journal·2003·237 cit.·DOI
10
Exosomal microRNA miR-1246 induces cell motility and invasion through the regulation of DENND2D in oral squamous cell carcinoma
Scientific Reports·2016·195 cit.·DOI

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