Cell Signaling Technology

CD79B (D2N3H) Rabbit Monoclonal Antibody (BSA and Azide Free)

#24566 · $865

31
Citing papers
1.1k
Combined citations
Supplier page Find this product

Research areas

Alzheimer's disease research and treatments12Autophagy in Disease and Therapy6Traumatic Brain Injury and Neurovascular Disturbances4Neuroinflammation and Neurodegeneration Mechanisms4Neuroscience and Neuropharmacology Research3Endoplasmic Reticulum Stress and Disease3Medicinal Plants and Neuroprotection2Ion channel regulation and function2

Papers citing this product

1
Activation of PERK Elicits Memory Impairment through Inactivation of CREB and Downregulation of PSD95 After Traumatic Brain Injury
Journal of Neuroscience·2017·116 cit.·DOI
2
Metformin Ameliorates A<i>β</i> Pathology by Insulin-Degrading Enzyme in a Transgenic Mouse Model of Alzheimer’s Disease
Oxidative Medicine and Cellular Longevity·2020·105 cit.·DOI
3
MiR-299-5p regulates apoptosis through autophagy in neurons and ameliorates cognitive capacity in APPswe/PS1dE9 mice
Scientific Reports·2016·74 cit.·DOI
4
RhoA-ROCK Inhibition Reverses Synaptic Remodeling and Motor and Cognitive Deficits Caused by Traumatic Brain Injury
Scientific Reports·2017·73 cit.·DOI
5
Polarity of varicosity initiation in central neuron mechanosensation
The Journal of Cell Biology·2017·67 cit.·DOI
6
Macrophage-Mediated Degradation of β-Amyloid via an Apolipoprotein E Isoform-Dependent Mechanism
Journal of Neuroscience·2009·66 cit.·DOI
7
Statins induce insulin-degrading enzyme secretion from astrocytes via an autophagy-based unconventional secretory pathway
Molecular Neurodegeneration·2015·64 cit.·DOI
8
Oligomeric Amyloid-β Inhibits the Proteolytic Conversion of Brain-derived Neurotrophic Factor (BDNF), AMPA Receptor Trafficking, and Classical Conditioning
Journal of Biological Chemistry·2010·63 cit.·DOI
9
Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer’s disease
Scientific Reports·2017·61 cit.·DOI
10
Alpha-synuclein alters differently gene expression of Sirts, PARPs and other stress response proteins: implications for neurodegenerative disorders
Molecular Neurobiology·2017·49 cit.·DOI

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