Cell Signaling Technology

CrkL (32H4) Mouse Monoclonal Antibody

#3182 · $158,348

63

Evidence Score · Well-Established

Used by 123 independent labs across 132 papers. How we score this →

100

Citing papers

14.1k

Combined citations

Supplier page Find this product

Research areas

Cancer-related molecular mechanisms research8 MicroRNA in disease regulation6 Adipose Tissue and Metabolism6 RNA modifications and cancer5 PI3K/AKT/mTOR signaling in cancer5 RNA Research and Splicing5 Immune Cell Function and Interaction4 Circular RNAs in diseases4

Papers citing this product

1

A GPX4-dependent cancer cell state underlies the clear-cell morphology and confers sensitivity to ferroptosis

Nature Communications·2019·859 cit.·DOI

2

Post-transcriptional Regulation of Low Density Lipoprotein Receptor Protein by Proprotein Convertase Subtilisin/Kexin Type 9a in Mouse Liver

Journal of Biological Chemistry·2004·501 cit.·DOI

3

Psychedelics promote plasticity by directly binding to BDNF receptor TrkB

Nature Neuroscience·2023·421 cit.·DOI

4

Phosphorylation of the adaptor ASC acts as a molecular switch that controls the formation of speck-like aggregates and inflammasome activity

Nature Immunology·2013·378 cit.·DOI

5

Akt inhibitors in cancer treatment: The long journey from drug discovery to clinical use (Review)

International Journal of Oncology·2015·363 cit.·DOI

6

Exosomes derived from miR‐375‐overexpressing human adipose mesenchymal stem cells promote bone regeneration

Cell Proliferation·2019·358 cit.·DOI

7

RETRACTED: Breast Tumor Cells with PI3K Mutation or HER2 Amplification Are Selectively Addicted to Akt Signaling

PLoS ONE·2008·317 cit.·DOI

8

Central insulin action regulates peripheral glucose and fat metabolism in mice

Journal of Clinical Investigation·2008·267 cit.·DOI

9

Mice fed a lipogenic methionine-choline-deficient diet develop hypermetabolism coincident with hepatic suppression of SCD-1

Journal of Lipid Research·2006·261 cit.·DOI

10

CD91-dependent programming of T-helper cell responses following heat shock protein immunization

Nature Communications·2011·243 cit.·DOI

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