Cell Signaling Technology

PathScan® Phospho-Akt1 (Ser473) Sandwich ELISA Kit

#7160 · $661

100
Citing papers
14.2k
Combined citations
Supplier page Find this product

Research areas

Neurogenesis and neuroplasticity mechanisms12Endoplasmic Reticulum Stress and Disease11Epigenetics and DNA Methylation7Cancer Cells and Metastasis7Cytokine Signaling Pathways and Interactions7Axon Guidance and Neuronal Signaling6CRISPR and Genetic Engineering6Pancreatic function and diabetes5

Papers citing this product

1
Metformin inhibits hepatic gluconeogenesis in mice independently of the LKB1/AMPK pathway via a decrease in hepatic energy state
Journal of Clinical Investigation·2010·1.2k cit.·DOI
2
Endoplasmic reticulum stress contributes to beta cell apoptosis in type 2 diabetes
Diabetologia·2007·819 cit.·DOI
3
FGF stimulation of the Erk1/2 signalling cascade triggers transition of pluripotent embryonic stem cells from self-renewal to lineage commitment
Development·2007·805 cit.·DOI
4
RETRACTED ARTICLE: A mechanism for the suppression of homologous recombination in G1 cells
Nature·2015·476 cit.·DOI
5
Cancer-associated fibroblasts regulate the plasticity of lung cancer stemness via paracrine signalling
Nature Communications·2014·441 cit.·DOI
6
Variability of doublecortin-associated dendrite maturation in adult hippocampal neurogenesis is independent of the regulation of precursor cell proliferation
BMC Neuroscience·2006·392 cit.·DOI
7
Decreased adult hippocampal neurogenesis in the PDAPP mouse model of Alzheimer's disease
The Journal of Comparative Neurology·2006·353 cit.·DOI
8
Activin A Maintains Self-Renewal and Regulates Fibroblast Growth Factor, Wnt, and Bone Morphogenic Protein Pathways in Human Embryonic Stem Cells
Stem Cells·2006·351 cit.·DOI
9
Inhibiting the system xC−/glutathione axis selectively targets cancers with mutant-p53 accumulation
Nature Communications·2017·321 cit.·DOI
10
Mesenchymal Stem Cells Secrete Multiple Cytokines That Promote Angiogenesis and Have Contrasting Effects on Chemotaxis and Apoptosis
PLoS ONE·2012·305 cit.·DOI

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