Cell Signaling Technology

Phospho-NDRG1 (Thr346) (D98G11) Rabbit Monoclonal Antibody (HRP Conjugate)

#8947 · $493

98

Citing papers

2.7k

Combined citations

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Research areas

Cancer Cells and Metastasis7 MicroRNA in disease regulation7 Immunotherapy and Immune Responses6 PI3K/AKT/mTOR signaling in cancer5 Cancer, Hypoxia, and Metabolism5 Parkinson's Disease Mechanisms and Treatments5 NF-κB Signaling Pathways5 RNA Research and Splicing5

Papers citing this product

1

Phosphatidylinositol 3-Kinase Activates ERK in Primary Sensory Neurons and Mediates Inflammatory Heat Hyperalgesia through TRPV1 Sensitization

Journal of Neuroscience·2004·415 cit.·DOI

2

Extracellular caspase-6 drives murine inflammatory pain via microglial TNF-α secretion

Journal of Clinical Investigation·2014·193 cit.·DOI

3

Prion Protein-mediated Toxicity of Amyloid-β Oligomers Requires Lipid Rafts and the Transmembrane LRP1

Journal of Biological Chemistry·2013·150 cit.·DOI

4

Protein kinase D1 drives pancreatic acinar cell reprogramming and progression to intraepithelial neoplasia

Nature Communications·2015·101 cit.·DOI

5

Chlorogenic Acid Inhibits Liver Fibrosis by Blocking the miR-21-Regulated TGF-β1/Smad7 Signaling Pathway in Vitro and in Vivo

Frontiers in Pharmacology·2017·100 cit.·DOI

6

Novel protein kinase D inhibitors cause potent arrest in prostate cancer cell growth and motility

BMC Chemical Biology·2010·85 cit.·DOI

7

Differentiation by NK cells is a prerequisite for effective targeting of cancer stem cells/poorly differentiated tumors by chemopreventive and chemotherapeutic drugs

Journal of Cancer·2017·75 cit.·DOI

8

The role of the cancer stem cell marker CD271 in DNA damage response and drug resistance of melanoma cells

Oncogenesis·2017·65 cit.·DOI

9

Protein Kinase D Regulates Cell Death Pathways in Experimental Pancreatitis

Frontiers in Physiology·2012·61 cit.·DOI

10

Neuropilin-1 promotes the oncogenic Tenascin-C/integrin β3 pathway and modulates chemoresistance in breast cancer cells

BMC Cancer·2018·59 cit.·DOI

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