Cell Signaling Technology

IRE1 alpha (14C10) Rabbit Monoclonal Antibody

#3294 · western blot · $152,844

100

Citing papers

14.2k

Combined citations

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Research areas

Endoplasmic Reticulum Stress and Disease20 Genomics, phytochemicals, and oxidative stress7 Autophagy in Disease and Therapy6 Glycosylation and Glycoproteins Research5 Cytokine Signaling Pathways and Interactions5 RNA regulation and disease5 Estrogen and related hormone effects4 Cancer Cells and Metastasis4

Papers citing this product

1

Dietary tryptophan metabolite released by intratumoral Lactobacillus reuteri facilitates immune checkpoint inhibitor treatment

Cell·2023·635 cit.·DOI

2

Rapid Akt activation by nicotine and a tobacco carcinogen modulates the phenotype of normal human airway epithelial cells

Journal of Clinical Investigation·2003·511 cit.·DOI

3

The PTEN-regulating microRNA miR-26a is amplified in high-grade glioma and facilitates gliomagenesis in vivo

Genes & Development·2009·509 cit.·DOI

4

Rapid Akt activation by nicotine and a tobacco carcinogen modulates the phenotype of normal human airway epithelial cells

Journal of Clinical Investigation·2003·500 cit.·DOI

5

IL-6/JAK1 pathway drives PD-L1 Y112 phosphorylation to promote cancer immune evasion

Journal of Clinical Investigation·2019·380 cit.·DOI

6

O-GlcNAcylation of PGK1 coordinates glycolysis and TCA cycle to promote tumor growth

Nature Communications·2020·345 cit.·DOI

7

LEF-1 and TCF-1 orchestrate TFH differentiation by regulating differentiation circuits upstream of the transcriptional repressor Bcl6

Nature Immunology·2015·326 cit.·DOI

8

Tobacco components stimulate Akt-dependent proliferation and NFκB-dependent survival in lung cancer cells

Carcinogenesis·2005·289 cit.·DOI

9

Pharmacological targeting of MYC-regulated IRE1/XBP1 pathway suppresses MYC-driven breast cancer

Journal of Clinical Investigation·2018·260 cit.·DOI

10

Nicotine Inactivation of the Proapoptotic Function of Bax through Phosphorylation

Journal of Biological Chemistry·2005·252 cit.·DOI

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