Cell Signaling Technology

TRIF (F6K4E) Rabbit Monoclonal Antibody

#16809 · western blot · $161,376

63
Evidence Score · Well-Established
Used by 131 independent labs across 140 papers. How we score this →
40
Citing papers
2.6k
Combined citations
Supplier page Find this product

Research areas

Mitochondrial Function and Pathology6Adipose Tissue and Metabolism5Cholesterol and Lipid Metabolism5Protein Kinase Regulation and GTPase Signaling3Melanoma and MAPK Pathways3Lipid metabolism and biosynthesis3Metabolism and Genetic Disorders3Computational Drug Discovery Methods2

Papers citing this product

1
Longitudinal observation and decline of neutralizing antibody responses in the three months following SARS-CoV-2 infection in humans
Nature Microbiology·2020·1.4k cit.·DOI
2
Cytosolic ROS production by NADPH oxidase 2 regulates muscle glucose uptake during exercise
Nature Communications·2019·213 cit.·DOI
3
C-Jun N-terminal kinase controls TDP-43 accumulation in stress granules induced by oxidative stress
Molecular Neurodegeneration·2011·109 cit.·DOI
4
Xanthohumol Improves Diet-induced Obesity and Fatty Liver by Suppressing Sterol Regulatory Element-binding Protein (SREBP) Activation
Journal of Biological Chemistry·2015·102 cit.·DOI
5
Mitochondrial PE potentiates respiratory enzymes to amplify skeletal muscle aerobic capacity
Science Advances·2019·100 cit.·DOI
6
Nerve Injury-Induced Chronic Pain Is Associated with Persistent DNA Methylation Reprogramming in Dorsal Root Ganglion
Journal of Neuroscience·2018·95 cit.·DOI
7
Growth Factor-induced Phosphorylation of Sterol Regulatory Element-binding Proteins Inhibits Sumoylation, Thereby Stimulating the Expression of Their Target Genes, Low Density Lipoprotein Uptake, and Lipid Synthesis
Journal of Biological Chemistry·2008·79 cit.·DOI
8
Identification of a Novel Transport-independent Function of PiT1/SLC20A1 in the Regulation of TNF-induced Apoptosis
Journal of Biological Chemistry·2010·69 cit.·DOI
9
Critical role of c-jun N-terminal protein kinase in promoting mitochondrial dysfunction and acute liver injury
Redox Biology·2015·49 cit.·DOI
10
ETHE1 and MOCS1 deficiencies: Disruption of mitochondrial bioenergetics, dynamics, redox homeostasis and endoplasmic reticulum-mitochondria crosstalk in patient fibroblasts
Scientific Reports·2019·41 cit.·DOI

Similar products

ProductVendorEvidencePricePapers
TRIF AntibodyRabbitCell Signaling Technology63$317100

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