Cell Signaling Technology

SimpleChIP® Mouse Oct-4 Promoter Primers

#4653 · $137

100
Citing papers
10.8k
Combined citations
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Research areas

MicroRNA in disease regulation23Circular RNAs in diseases17Cancer-related molecular mechanisms research16RNA Research and Splicing8Immune Cell Function and Interaction7Immune Response and Inflammation5Cytokine Signaling Pathways and Interactions5Immunotherapy and Immune Responses5

Papers citing this product

1
Tumor-suppressive <i>miR-34a</i> induces senescence-like growth arrest through modulation of the E2F pathway in human colon cancer cells
Proceedings of the National Academy of Sciences·2007·902 cit.·DOI
2
RSL3 Drives Ferroptosis Through GPX4 Inactivation and ROS Production in Colorectal Cancer
Frontiers in Pharmacology·2018·694 cit.·DOI
3
B cell-derived GABA elicits IL-10+ macrophages to limit anti-tumour immunity
Nature·2021·428 cit.·DOI
4
Targeting Interleukin 6 Signaling Suppresses Glioma Stem Cell Survival and Tumor Growth
Stem Cells·2009·336 cit.·DOI
5
ATP Inhibits the Generation and Function of Regulatory T Cells Through the Activation of Purinergic P2X Receptors
Science Signaling·2011·284 cit.·DOI
6
Isl1Cre reveals a common Bmp pathway in heart and limb development
Development·2006·269 cit.·DOI
7
MicroRNA-338 Regulates Local Cytochrome<i>c</i>Oxidase IV mRNA Levels and Oxidative Phosphorylation in the Axons of Sympathetic Neurons
Journal of Neuroscience·2008·254 cit.·DOI
8
Macrophage-secreted IL-8 induces epithelial-mesenchymal transition in hepatocellular carcinoma cells by activating the JAK2/STAT3/Snail pathway
International Journal of Oncology·2014·228 cit.·DOI
9
JAK1 Activates STAT3 Activity in Non-Small–Cell Lung Cancer Cells and IL-6 Neutralizing Antibodies Can Suppress JAK1-STAT3 Signaling
Molecular Cancer Therapeutics·2011·180 cit.·DOI
10
Berberine prevents hyperglycemia-induced endothelial injury and enhances vasodilatation via adenosine monophosphate-activated protein kinase and endothelial nitric oxide synthase
Cardiovascular Research·2009·161 cit.·DOI