Cell Signaling Technology

KLF2 (E2Z1H) Rabbit Monoclonal Antibody

#15306 · $161

53
Citing papers
1.9k
Combined citations
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Research areas

Adipose Tissue and Metabolism7Muscle Physiology and Disorders7Mitochondrial Function and Pathology6Cell Adhesion Molecules Research5Regulation of Appetite and Obesity5Cancer, Hypoxia, and Metabolism4ATP Synthase and ATPases Research4Cardiomyopathy and Myosin Studies4

Papers citing this product

1
Individual cristae within the same mitochondrion display different membrane potentials and are functionally independent
The EMBO Journal·2019·323 cit.·DOI
2
Accurate Recycling of Parental Histones Reproduces the Histone Modification Landscape during DNA Replication
Molecular Cell·2018·263 cit.·DOI
3
Quercetin Suppresses the Progression of Atherosclerosis by Regulating MST1-Mediated Autophagy in ox-LDL-Induced RAW264.7 Macrophage Foam Cells
International Journal of Molecular Sciences·2019·176 cit.·DOI
4
Glutamatergic Preoptic Area Neurons That Express Leptin Receptors Drive Temperature-Dependent Body Weight Homeostasis
Journal of Neuroscience·2016·161 cit.·DOI
5
Oxytocin-receptor-expressing neurons in the parabrachial nucleus regulate fluid intake
Nature Neuroscience·2017·116 cit.·DOI
6
Transforming Growth Factor-β Promotes Survival of Mammary Carcinoma Cells through Induction of Antiapoptotic Transcription Factor DEC1
Cancer Research·2007·91 cit.·DOI
7
Leptin Stimulates Neuropeptide Y and Cocaine Amphetamine-Regulated Transcript Coexpressing Neuronal Activity in the Dorsomedial Hypothalamus in Diet-Induced Obese Mice
Journal of Neuroscience·2013·75 cit.·DOI
8
Inhibition of ATP synthase reverse activity restores energy homeostasis in mitochondrial pathologies
The EMBO Journal·2023·65 cit.·DOI
9
Differentiated embryonic chondrocyte-expressed gene 1 is associated with hypoxia-inducible factor 1α and Ki67 in human gastric cancer
Diagnostic Pathology·2013·44 cit.·DOI
10
Membralin deficiency dysregulates astrocytic glutamate homeostasis, leading to ALS-like impairment
Journal of Clinical Investigation·2019·44 cit.·DOI

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