Cell Signaling Technology

N-Myc (D4F9Z) Rabbit Monoclonal Antibody (PE Conjugate)

#17150 · $465

28
Citing papers
562
Combined citations
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Research areas

Neuroscience and Neuropharmacology Research3Cholesterol and Lipid Metabolism2Autophagy in Disease and Therapy2Cancer Genomics and Diagnostics2Adipose Tissue and Metabolism2Cellular Mechanics and Interactions2Axon Guidance and Neuronal Signaling2Cancer Research and Treatments2

Papers citing this product

1
MiR-155 modulates the inflammatory phenotype of intestinal myofibroblasts by targeting SOCS1 in ulcerative colitis
Experimental & Molecular Medicine·2015·120 cit.·DOI
2
Overexpression of DeltaFosB in nucleus accumbens mimics the protective addiction phenotype, but not the protective depression phenotype of environmental enrichment
Frontiers in Behavioral Neuroscience·2014·57 cit.·DOI
3
<p>EMP1 Promotes the Proliferation and Invasion of Ovarian Cancer Cells Through Activating the MAPK Pathway</p>
OncoTargets and Therapy·2020·55 cit.·DOI
4
The Interactions of Hepatocyte Growth Factor/Scatter Factor and Its NK1 and NK2 Variants with Glycosaminoglycans Using a Modified Gel Mobility Shift Assay
Journal of Biological Chemistry·2004·54 cit.·DOI
5
CFTR-regulated MAPK/NF-κB signaling in pulmonary inflammation in thermal inhalation injury
Scientific Reports·2015·47 cit.·DOI
6
β-Amyloid Inhibits Protein Prenylation and Induces Cholesterol Sequestration by Impairing SREBP-2 Cleavage
Journal of Neuroscience·2012·40 cit.·DOI
7
Folic Acid-Modified Fluorescent-Magnetic Nanoparticles for Efficient Isolation and Identification of Circulating Tumor Cells in Ovarian Cancer
Biosensors·2022·30 cit.·DOI
8
A central role of IKK2 and TPL2 in JNK activation and viral B-cell transformation
Nature Communications·2020·29 cit.·DOI
9
Dominant-negative ATF5 rapidly depletes survivin in tumor cells
Cell Death and Disease·2019·22 cit.·DOI
10
MiR-135b-5p is an oncogene in pancreatic cancer to regulate GPRC5A expression by targeting transcription factor KLF4
Cell Death Discovery·2022·21 cit.·DOI

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