Cell Signaling Technology

Phospho-C/EBP beta (Thr235) Antibody

#3084 · western blot · $148,362

100
Citing papers
13.2k
Combined citations
Supplier page Find this product

Research areas

Endoplasmic Reticulum Stress and Disease11Metabolism, Diabetes, and Cancer8Immune cells in cancer8MicroRNA in disease regulation7Pancreatic function and diabetes6Cancer Cells and Metastasis5Epigenetics and DNA Methylation5Immune Cell Function and Interaction5

Papers citing this product

1
FGF stimulation of the Erk1/2 signalling cascade triggers transition of pluripotent embryonic stem cells from self-renewal to lineage commitment
Development·2007·805 cit.·DOI
2
SOCS3 Deficiency Promotes M1 Macrophage Polarization and Inflammation
The Journal of Immunology·2012·464 cit.·DOI
3
Antitumor activity of MLN8054, an orally active small-molecule inhibitor of Aurora A kinase
Proceedings of the National Academy of Sciences·2007·413 cit.·DOI
4
Inhibition of Insulin Sensitivity by Free Fatty Acids Requires Activation of Multiple Serine Kinases in 3T3-L1 Adipocytes
Molecular Endocrinology·2004·331 cit.·DOI
5
Functional exosome-mediated co-delivery of doxorubicin and hydrophobically modified microRNA 159 for triple-negative breast cancer therapy
Journal of Nanobiotechnology·2019·328 cit.·DOI
6
Tyrosine Phosphorylation of VE-cadherin Prevents Binding of p120- and β-Catenin and Maintains the Cellular Mesenchymal State
Journal of Biological Chemistry·2005·325 cit.·DOI
7
IL-6 and IL-10 Anti-Inflammatory Activity Links Exercise to Hypothalamic Insulin and Leptin Sensitivity through IKKβ and ER Stress Inhibition
PLoS Biology·2010·324 cit.·DOI
8
Microbial metabolite butyrate facilitates M2 macrophage polarization and function
Scientific Reports·2016·313 cit.·DOI
9
Activation of the Akt-NF-κB Pathway by Subtilase Cytotoxin through the ATF6 Branch of the Unfolded Protein Response
The Journal of Immunology·2009·283 cit.·DOI
10
Complementary roles of IRS-1 and IRS-2 in the hepatic regulation of metabolism
Journal of Clinical Investigation·2005·273 cit.·DOI

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