Cell Signaling Technology

CX3CR1 (E9J4I) Rabbit Monoclonal Antibody

#19771 · $161,376

61
Evidence Score · Well-Established
Used by 87 independent labs across 100 papers. How we score this →
100
Citing papers
2.9k
Combined citations
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Research areas

Inflammasome and immune disorders38Heme Oxygenase-1 and Carbon Monoxide8Autophagy in Disease and Therapy7Cellular Mechanics and Interactions6interferon and immune responses6Microtubule and mitosis dynamics5Neuroinflammation and Neurodegeneration Mechanisms5Cancer-related Molecular Pathways4

Papers citing this product

1
Mutation in TECPR2 Reveals a Role for Autophagy in Hereditary Spastic Paraparesis
The American Journal of Human Genetics·2012·167 cit.·DOI
2
Fucoidan Extract Induces Apoptosis in MCF-7 Cells via a Mechanism Involving the ROS-Dependent JNK Activation and Mitochondria-Mediated Pathways
PLoS ONE·2011·166 cit.·DOI
3
TRIM28 SUMOylates and stabilizes NLRP3 to facilitate inflammasome activation
Nature Communications·2021·156 cit.·DOI
4
Protection of Mcc950 against high-glucose-induced human retinal endothelial cell dysfunction
Cell Death and Disease·2017·138 cit.·DOI
5
Salidroside Attenuates High‐Fat Diet‐Induced Nonalcoholic Fatty Liver Disease via AMPK‐Dependent TXNIP/NLRP3 Pathway
Oxidative Medicine and Cellular Longevity·2018·130 cit.·DOI
6
Exosomal TRIM3 is a novel marker and therapy target for gastric cancer
Journal of Experimental & Clinical Cancer Research·2018·129 cit.·DOI
7
Enucleated cells reveal differential roles of the nucleus in cell migration, polarity, and mechanotransduction
The Journal of Cell Biology·2018·121 cit.·DOI
8
Dl-3-n-Butylphthalide Rescues Dopaminergic Neurons in Parkinson’s Disease Models by Inhibiting the NLRP3 Inflammasome and Ameliorating Mitochondrial Impairment
Frontiers in Immunology·2021·110 cit.·DOI
9
Melanoblast transcriptome analysis reveals pathways promoting melanoma metastasis
Nature Communications·2020·95 cit.·DOI
10
Mucin O-glycan-microbiota axis orchestrates gut homeostasis in a diarrheal pig model
Microbiome·2022·86 cit.·DOI

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