Cell Signaling Technology

Akt (pan) (C67E7) Rabbit Monoclonal Antibody (Biotinylated)

#5373 · $556

67
Evidence Score · Well-Established
Used by 2,346 independent labs across 2,626 papers. How we score this →
100
Citing papers
10.2k
Combined citations
Supplier page Find this product

Research areas

Cancer Cells and Metastasis10Immune cells in cancer8Epigenetics and DNA Methylation8Immune Response and Inflammation7PI3K/AKT/mTOR signaling in cancer5Cytokine Signaling Pathways and Interactions4Cancer, Hypoxia, and Metabolism4Immune Cell Function and Interaction4

Papers citing this product

1
The matrix component biglycan is proinflammatory and signals through Toll-like receptors 4 and 2 in macrophages
Journal of Clinical Investigation·2005·836 cit.·DOI
2
P2X7-Dependent Release of Interleukin-1β and Nociception in the Spinal Cord following Lipopolysaccharide
Journal of Neuroscience·2010·292 cit.·DOI
3
The tale of TILs in breast cancer: A report from The International Immuno-Oncology Biomarker Working Group
npj Breast Cancer·2021·269 cit.·DOI
4
ERK1/2 Phosphorylate Raptor to Promote Ras-dependent Activation of mTOR Complex 1 (mTORC1)
Journal of Biological Chemistry·2010·266 cit.·DOI
5
Stress Acts Cumulatively To Precipitate Alzheimer's Disease-Like Tau Pathology and Cognitive Deficits
Journal of Neuroscience·2011·248 cit.·DOI
6
UCP2-induced fatty acid synthase promotes NLRP3 inflammasome activation during sepsis
Journal of Clinical Investigation·2015·239 cit.·DOI
7
Notch signaling is activated by TLR stimulation and regulates macrophage functions
European Journal of Immunology·2007·232 cit.·DOI
8
DOT1L cooperates with the c-Myc-p300 complex to epigenetically derepress CDH1 transcription factors in breast cancer progression
Nature Communications·2015·229 cit.·DOI
9
Macrophage-cancer hybrid membrane-coated nanoparticles for targeting lung metastasis in breast cancer therapy
Journal of Nanobiotechnology·2020·199 cit.·DOI
10
The APC/C inhibitor, Emi1, is essential for prevention of rereplication
Genes & Development·2007·199 cit.·DOI

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