Cell Signaling Technology

SignalSlide® Phospho-Akt (Ser473) IHC Controls

#8101 · immunohistochemistry · $196

100

Citing papers

7.8k

Combined citations

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Research areas

PI3K/AKT/mTOR signaling in cancer10 Chemokine receptors and signaling9 Immunotherapy and Immune Responses9 Protein Tyrosine Phosphatases7 Immune Cell Function and Interaction7 Immune Response and Inflammation7 interferon and immune responses7 NF-κB Signaling Pathways6

Papers citing this product

1

Suppression of insulin feedback enhances the efficacy of PI3K inhibitors

Nature·2018·706 cit.·DOI

2

Ribosomal protein S6 phosphorylation is a determinant of cell size and glucose homeostasis

Genes & Development·2005·636 cit.·DOI

3

Enhancing CAR T cell persistence through ICOS and 4-1BB costimulation

JCI Insight·2018·573 cit.·DOI

4

Iterative Design and Optimization of Initially Inactive Proteolysis Targeting Chimeras (PROTACs) Identify VZ185 as a Potent, Fast, and Selective von Hippel–Lindau (VHL) Based Dual Degrader Probe of BRD9 and BRD7

Journal of Medicinal Chemistry·2018·330 cit.·DOI

5

BRD9 defines a SWI/SNF sub-complex and constitutes a specific vulnerability in malignant rhabdoid tumors

Nature Communications·2019·216 cit.·DOI

6

PSGL-1 Is an Immune Checkpoint Regulator that Promotes T Cell Exhaustion

Immunity·2016·173 cit.·DOI

7

Mitogen-Activated Protein Kinase Upregulates the Dendritic Translation Machinery in Long-Term Potentiation by Controlling the Mammalian Target of Rapamycin Pathway

Journal of Neuroscience·2007·170 cit.·DOI

8

TRPV1 Activation Is Required for Hypertonicity-Stimulated Inflammatory Cytokine Release in Human Corneal Epithelial Cells

Investigative Ophthalmology & Visual Science·2010·155 cit.·DOI

9

Anionic Pulmonary Surfactant Phospholipids Inhibit Inflammatory Responses from Alveolar Macrophages and U937 Cells by Binding the Lipopolysaccharide-interacting Proteins CD14 and MD-2

Journal of Biological Chemistry·2009·147 cit.·DOI

10

4-1BB enhancement of CAR T function requires NF-κB and TRAFs

JCI Insight·2018·147 cit.·DOI

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