Cell Signaling Technology

HMGA1 (D4F8) Rabbit Monoclonal Antibody

#12094 · $161

79
Citing papers
1.7k
Combined citations
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Research areas

Receptor Mechanisms and Signaling6Neuropeptides and Animal Physiology5RNA modifications and cancer5RNA Interference and Gene Delivery5MicroRNA in disease regulation5Microtubule and mitosis dynamics4Pancreatic function and diabetes4RNA Research and Splicing4

Papers citing this product

1
NBK/BIK antagonizes MCL-1 and BCL-X<sub>L</sub> and activates BAK-mediated apoptosis in response to protein synthesis inhibition
Genes & Development·2007·134 cit.·DOI
2
Therapeutic Targeting of Th17/Tc17 Cells Leads to Clinical Improvement of Lichen Planus
Frontiers in Immunology·2019·115 cit.·DOI
3
Activation of CREB in the Nucleus Accumbens Shell Produces Anhedonia and Resistance to Extinction of Fear in Rats
Journal of Neuroscience·2011·100 cit.·DOI
4
Protective Effects of Short-Chain Fatty Acids on Endothelial Dysfunction Induced by Angiotensin II
Frontiers in Physiology·2020·99 cit.·DOI
5
Selective autophagy maintains centrosome integrity and accurate mitosis by turnover of centriolar satellites
Nature Communications·2019·84 cit.·DOI
6
Cdc42 Regulates Arsenic-induced NADPH Oxidase Activation and Cell Migration through Actin Filament Reorganization
Journal of Biological Chemistry·2004·84 cit.·DOI
7
Hyperglycemia-Induced Oxidative Stress Abrogates Remifentanil Preconditioning-Mediated Cardioprotection in Diabetic Rats by Impairing Caveolin-3-Modulated PI3K/Akt and JAK2/STAT3 Signaling
Oxidative Medicine and Cellular Longevity·2019·66 cit.·DOI
8
Triphala Extract Suppresses Proliferation and Induces Apoptosis in Human Colon Cancer Stem Cells via Suppressing c-Myc/Cyclin D1 and Elevation of Bax/Bcl-2 Ratio
BioMed Research International·2015·66 cit.·DOI
9
N-acetylcysteine attenuates myocardial dysfunction and postischemic injury by restoring caveolin-3/eNOS signaling in diabetic rats
Cardiovascular Diabetology·2016·60 cit.·DOI
10
A viral ADP-ribosyltransferase attaches RNA chains to host proteins
Nature·2023·46 cit.·DOI

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