Cell Signaling Technology

PDK1 (D4Q4D) Rabbit Monoclonal Antibody

#13037 · $161

85
Citing papers
3.4k
Combined citations
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Research areas

Ubiquitin and proteasome pathways8Autophagy in Disease and Therapy7Ion channel regulation and function6Alzheimer's disease research and treatments6Cancer-related molecular mechanisms research5Histone Deacetylase Inhibitors Research4RNA Research and Splicing4Hepatitis C virus research4

Papers citing this product

1
A novel tumor suppressor protein encoded by circular AKT3 RNA inhibits glioblastoma tumorigenicity by competing with active phosphoinositide-dependent Kinase-1
Molecular Cancer·2019·406 cit.·DOI
2
Hypersensitivity to mGluR5 and ERK1/2 Leads to Excessive Protein Synthesis in the Hippocampus of a Mouse Model of Fragile X Syndrome
Journal of Neuroscience·2010·384 cit.·DOI
3
Cytochrome P450 2E1 contributes to ethanol-induced fatty liver in mice
Hepatology·2008·304 cit.·DOI
4
Regulation of Eukaryotic Initiation Factor 4E by Converging Signaling Pathways during Metabotropic Glutamate Receptor-Dependent Long-Term Depression
Journal of Neuroscience·2006·244 cit.·DOI
5
A Novel RNA-Binding Protein in Neuronal RNA Granules: Regulatory Machinery for Local Translation
Journal of Neuroscience·2005·114 cit.·DOI
6
The gut microbiota regulates hypothalamic inflammation and leptin sensitivity in Western diet-fed mice via a GLP-1R-dependent mechanism
Cell Reports·2021·112 cit.·DOI
7
Fibrin accumulation plays a critical role in the sensitization to lipopolysaccharide-induced liver injury caused by ethanol in mice #
Hepatology·2009·92 cit.·DOI
8
PRMT5-mediated arginine methylation activates AKT kinase to govern tumorigenesis
Nature Communications·2021·86 cit.·DOI
9
Actinin-4 Governs Dendritic Spine Dynamics and Promotes Their Remodeling by Metabotropic Glutamate Receptors
Journal of Biological Chemistry·2015·82 cit.·DOI
10
Glabridin Prevents Doxorubicin-Induced Cardiotoxicity Through Gut Microbiota Modulation and Colonic Macrophage Polarization in Mice
Frontiers in Pharmacology·2019·75 cit.·DOI

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