Cell Signaling Technology

FastScan™ Total TBK1/NAK ELISA Kit

#15816 · $678

73
Citing papers
3.1k
Combined citations
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Research areas

Tuberous Sclerosis Complex Research12Drug Transport and Resistance Mechanisms11Endoplasmic Reticulum Stress and Disease5PI3K/AKT/mTOR signaling in cancer5Hippo pathway signaling and YAP/TAZ5Epigenetics and DNA Methylation4Liver physiology and pathology4Fibroblast Growth Factor Research4

Papers citing this product

1
Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesis
Genes & Development·2003·681 cit.·DOI
2
Bile Acids Induce Inflammatory Genes in Hepatocytes
American Journal Of Pathology·2011·496 cit.·DOI
3
Fluid shear stress activates YAP1 to promote cancer cell motility
Nature Communications·2017·259 cit.·DOI
4
Regulation of Bile Acid Synthesis by the Nuclear Receptor Rev-erbα
Gastroenterology·2008·201 cit.·DOI
5
Interleukin-1β-mediated Suppression of RXR:RAR Transactivation of the Ntcp Promoter Is JNK-dependent
Journal of Biological Chemistry·2002·119 cit.·DOI
6
FGF15/FGFR4 Integrates Growth Factor Signaling with Hepatic Bile Acid Metabolism and Insulin Action
Journal of Biological Chemistry·2009·112 cit.·DOI
7
Independent Repression of Bile Acid Synthesis and Activation of c-Jun N-terminal Kinase (JNK) by Activated Hepatocyte Fibroblast Growth Factor Receptor 4 (FGFR4) and Bile Acids
Journal of Biological Chemistry·2005·102 cit.·DOI
8
Biallelic Mutations in<i>TSC2</i>Lead to Abnormalities Associated with Cortical Tubers in Human iPSC-Derived Neurons
Journal of Neuroscience·2019·100 cit.·DOI
9
Subependymal giant cell astrocytomas in Tuberous Sclerosis Complex have consistent <i>TSC1/TSC2</i> biallelic inactivation, and no <i>BRAF</i> mutations
Oncotarget·2017·76 cit.·DOI
10
A chronic high-cholesterol diet paradoxically suppresses hepatic CYP7A1 expression in FVB/NJ mice
Journal of Lipid Research·2010·63 cit.·DOI
FastScan™ Total TBK1/NAK ELISA Kit — Evidence — PubIndex