Cell Signaling Technology

FastScan™ Total TBK1/NAK ELISA Kit

#15816 · elisa · $67,830,490

73

Citing papers

3.1k

Combined citations

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Research areas

Tuberous Sclerosis Complex Research12 Drug Transport and Resistance Mechanisms11 Endoplasmic Reticulum Stress and Disease5 PI3K/AKT/mTOR signaling in cancer5 Hippo pathway signaling and YAP/TAZ5 Epigenetics and DNA Methylation4 Liver physiology and pathology4 Fibroblast Growth Factor Research4

Papers citing this product

1

Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesis

Genes & Development·2003·681 cit.·DOI

2

Bile Acids Induce Inflammatory Genes in Hepatocytes

American Journal Of Pathology·2011·496 cit.·DOI

3

Fluid shear stress activates YAP1 to promote cancer cell motility

Nature Communications·2017·259 cit.·DOI

4

Regulation of Bile Acid Synthesis by the Nuclear Receptor Rev-erbα

Gastroenterology·2008·201 cit.·DOI

5

Interleukin-1β-mediated Suppression of RXR:RAR Transactivation of the Ntcp Promoter Is JNK-dependent

Journal of Biological Chemistry·2002·119 cit.·DOI

6

FGF15/FGFR4 Integrates Growth Factor Signaling with Hepatic Bile Acid Metabolism and Insulin Action

Journal of Biological Chemistry·2009·112 cit.·DOI

7

Independent Repression of Bile Acid Synthesis and Activation of c-Jun N-terminal Kinase (JNK) by Activated Hepatocyte Fibroblast Growth Factor Receptor 4 (FGFR4) and Bile Acids

Journal of Biological Chemistry·2005·102 cit.·DOI

8

Biallelic Mutations in<i>TSC2</i>Lead to Abnormalities Associated with Cortical Tubers in Human iPSC-Derived Neurons

Journal of Neuroscience·2019·100 cit.·DOI

9

Subependymal giant cell astrocytomas in Tuberous Sclerosis Complex have consistent <i>TSC1/TSC2</i> biallelic inactivation, and no <i>BRAF</i> mutations

Oncotarget·2017·76 cit.·DOI

10

A chronic high-cholesterol diet paradoxically suppresses hepatic CYP7A1 expression in FVB/NJ mice

Journal of Lipid Research·2010·63 cit.·DOI

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