Cell Signaling Technology

Lamin B2 (E1S1Q) Rabbit Monoclonal Antibody

#13823 · $161

84
Citing papers
3.4k
Combined citations
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Research areas

Cardiac Fibrosis and Remodeling7Neuroinflammation and Neurodegeneration Mechanisms6Estrogen and related hormone effects6Histone Deacetylase Inhibitors Research6Menopause: Health Impacts and Treatments5Adipose Tissue and Metabolism5Muscle Physiology and Disorders5Nuclear Receptors and Signaling5

Papers citing this product

1
High-efficiency optogenetic silencing with soma-targeted anion-conducting channelrhodopsins
Nature Communications·2018·331 cit.·DOI
2
Estrogen Attenuates Ischemic Oxidative Damage via an Estrogen Receptor α-Mediated Inhibition of NADPH Oxidase Activation
Journal of Neuroscience·2009·241 cit.·DOI
3
The Blimp1–Bcl6 axis is critical to regulate osteoclast differentiation and bone homeostasis
The Journal of Experimental Medicine·2010·199 cit.·DOI
4
The ubiquitin E3 ligase TRAF6 exacerbates pathological cardiac hypertrophy via TAK1-dependent signalling
Nature Communications·2016·182 cit.·DOI
5
Extranuclear Estrogen Receptors Mediate the Neuroprotective Effects of Estrogen in the Rat Hippocampus
PLoS ONE·2010·163 cit.·DOI
6
MicroRNA-199a is induced in dystrophic muscle and affects WNT signaling, cell proliferation, and myogenic differentiation
Cell Death and Differentiation·2013·146 cit.·DOI
7
Role of Rac1 GTPase in NADPH Oxidase Activation and Cognitive Impairment Following Cerebral Ischemia in the Rat
PLoS ONE·2010·128 cit.·DOI
8
p53 induces miR199a-3p to suppress SOCS7 for STAT3 activation and renal fibrosis in UUO
Scientific Reports·2017·95 cit.·DOI
9
Neuroprotection from Retinal Ischemia/Reperfusion Injury by NOX2 NADPH Oxidase Deletion
Investigative Ophthalmology & Visual Science·2011·92 cit.·DOI
10
Activating Transcription Factor 3 Deficiency Promotes Cardiac Hypertrophy, Dysfunction, and Fibrosis Induced by Pressure Overload
PLoS ONE·2011·82 cit.·DOI

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