Cell Signaling Technology

DDX5 Antibody

#4387 · $317

100
Citing papers
8.6k
Combined citations
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Research areas

Cancer-related molecular mechanisms research12MicroRNA in disease regulation9Circular RNAs in diseases7Cancer, Hypoxia, and Metabolism6Autophagy in Disease and Therapy5Glycosylation and Glycoproteins Research5Cancer-related Molecular Pathways4Epigenetics and DNA Methylation4

Papers citing this product

1
Paracrine and Autocrine Regulation of Epidermal Growth Factor-Like Factors in Cumulus Oocyte Complexes and Granulosa Cells: Key Roles for Prostaglandin Synthase 2 and Progesterone Receptor
Molecular Endocrinology·2006·422 cit.·DOI
2
Thymic stromal lymphopoietin induces corticosteroid resistance in natural helper cells during airway inflammation
Nature Communications·2013·336 cit.·DOI
3
Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiency
Molecular Cell·2021·315 cit.·DOI
4
Gene Expression Profiles of Cumulus Cell Oocyte Complexes during Ovulation Reveal Cumulus Cells Express Neuronal and Immune-Related Genes: Does this Expand Their Role in the Ovulation Process?
Molecular Endocrinology·2006·264 cit.·DOI
5
Circular RNA hsa_circ_0004872 inhibits gastric cancer progression via the miR-224/Smad4/ADAR1 successive regulatory circuit
Molecular Cancer·2020·239 cit.·DOI
6
Neutrophils resist ferroptosis and promote breast cancer metastasis through aconitate decarboxylase 1
Cell Metabolism·2023·199 cit.·DOI
7
GPRC6A Mediates the Non-genomic Effects of Steroids
Journal of Biological Chemistry·2010·185 cit.·DOI
8
miR-29 targets Akt3 to reduce proliferation and facilitate differentiation of myoblasts in skeletal muscle development
Cell Death and Disease·2013·184 cit.·DOI
9
Hypoxia-induced autophagy drives colorectal cancer initiation and progression by activating the PRKC/PKC-EZR (ezrin) pathway
Autophagy·2019·183 cit.·DOI
10
Glioma-derived IL-33 orchestrates an inflammatory brain tumor microenvironment that accelerates glioma progression
Nature Communications·2020·179 cit.·DOI

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