Cell Signaling Technology

CDK4 (D9G3E) Rabbit Monoclonal Antibody (BSA and Azide Free)

#23972 · $865

20
Citing papers
396
Combined citations
Supplier page Find this product

Research areas

Autophagy in Disease and Therapy4Reproductive Biology and Fertility3Ubiquitin and proteasome pathways3Ovarian function and disorders2Genetics, Aging, and Longevity in Model Organisms2Protein Degradation and Inhibitors2Estrogen and related hormone effects2Multiple Myeloma Research and Treatments2

Papers citing this product

1
Linc00514 promotes breast cancer metastasis and M2 polarization of tumor-associated macrophages via Jagged1-mediated notch signaling pathway
Journal of Experimental & Clinical Cancer Research·2020·107 cit.·DOI
2
Rac1 modulates the formation of primordial follicles by facilitating STAT3-directed Jagged1, GDF9 and BMP15 transcription in mice
Scientific Reports·2016·68 cit.·DOI
3
A Gatekeeping Role of ESR2 to Maintain the Primordial Follicle Reserve
Endocrinology·2020·47 cit.·DOI
4
Inhibition of USP1 activates ER stress through Ubi-protein aggregation to induce autophagy and apoptosis in HCC
Cell Death and Disease·2022·41 cit.·DOI
5
Quantification and localization of oncogenic receptor tyrosine kinase variant transcripts using molecular inversion probes
Scientific Reports·2018·31 cit.·DOI
6
Comparative Cytotoxic Activity of Hydroxytyrosol and Its Semisynthetic Lipophilic Derivatives in Prostate Cancer Cells
Antioxidants·2021·20 cit.·DOI
7
Role of Lamin A/C as Candidate Biomarker of Aggressiveness and Tumorigenicity in Glioblastoma Multiforme
Biomedicines·2021·19 cit.·DOI
8
Silencing Autophagy-Related Gene 2 (ATG2) Results in Accelerated Senescence and Enhanced Immunity in Soybean
International Journal of Molecular Sciences·2021·16 cit.·DOI
9
PLEKHS1 drives PI3Ks and remodels pathway homeostasis in PTEN-null prostate
Molecular Cell·2023·15 cit.·DOI
10
Diosmin Promotes Myogenesis via Activating the Akt/FOXO1 Pathway to Facilitate the Proliferation of C2C12 Myoblasts
Journal of Agricultural and Food Chemistry·2023·11 cit.·DOI

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