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Cell Signaling Technology
Phospho-CD19 (Tyr531) Antibody
#3571 · $148
100
Citing papers
13.3k
Combined citations
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Research areas
Autophagy in Disease and Therapy
8
Epigenetics and DNA Methylation
7
PARP inhibition in cancer therapy
6
CRISPR and Genetic Engineering
6
DNA Repair Mechanisms
6
Immune Response and Inflammation
6
Mitochondrial Function and Pathology
6
Cell Adhesion Molecules Research
5
Papers citing this product
1
α-Synuclein Is Localized to Mitochondria-Associated ER Membranes
Journal of Neuroscience
·
2013
·
500 cit.
·
DOI
2
Cartilage Tissue Engineering by the 3D Bioprinting of iPS Cells in a Nanocellulose/Alginate Bioink
Scientific Reports
·
2017
·
475 cit.
·
DOI
3
Toll-like Receptors Induce a Phagocytic Gene Program through p38
The Journal of Experimental Medicine
·
2003
·
428 cit.
·
DOI
4
Differential Requirement for TANK-binding Kinase-1 in Type I Interferon Responses to Toll-like Receptor Activation and Viral Infection
The Journal of Experimental Medicine
·
2004
·
400 cit.
·
DOI
5
Radiosensitization and DNA repair inhibition by the combined use of novel inhibitors of DNA-dependent protein kinase and poly(ADP-ribose) polymerase-1.
PubMed
·
2003
·
331 cit.
6
Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiency
Molecular Cell
·
2021
·
315 cit.
·
DOI
7
Inferring and perturbing cell fate regulomes in human brain organoids
Nature
·
2022
·
309 cit.
·
DOI
8
VPS35 in Dopamine Neurons Is Required for Endosome-to-Golgi Retrieval of Lamp2a, a Receptor of Chaperone-Mediated Autophagy That Is Critical for -Synuclein Degradation and Prevention of Pathogenesis of Parkinson's Disease
Journal of Neuroscience
·
2015
·
270 cit.
·
DOI
9
Inhibition of Drp1 Ameliorates Synaptic Depression, Aβ Deposition, and Cognitive Impairment in an Alzheimer's Disease Model
Journal of Neuroscience
·
2017
·
257 cit.
·
DOI
10
Activation of the MAPK pathway is a common event in uveal melanomas although it rarely occurs through mutation of BRAF or RAS
British Journal of Cancer
·
2005
·
217 cit.
·
DOI
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