Cell Signaling Technology

Phospho-CD19 (Tyr531) Antibody

#3571 · $148,362

61

Evidence Score · Well-Established

Used by 97 independent labs across 100 papers. How we score this →

100

Citing papers

13.3k

Combined citations

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Research areas

Autophagy in Disease and Therapy8 Epigenetics and DNA Methylation7 PARP inhibition in cancer therapy6 CRISPR and Genetic Engineering6 DNA Repair Mechanisms6 Immune Response and Inflammation6 Mitochondrial Function and Pathology6 Cell Adhesion Molecules Research5

Papers citing this product

1

α-Synuclein Is Localized to Mitochondria-Associated ER Membranes

Journal of Neuroscience·2013·500 cit.·DOI

2

Cartilage Tissue Engineering by the 3D Bioprinting of iPS Cells in a Nanocellulose/Alginate Bioink

Scientific Reports·2017·475 cit.·DOI

3

Toll-like Receptors Induce a Phagocytic Gene Program through p38

The Journal of Experimental Medicine·2003·428 cit.·DOI

4

Differential Requirement for TANK-binding Kinase-1 in Type I Interferon Responses to Toll-like Receptor Activation and Viral Infection

The Journal of Experimental Medicine·2004·400 cit.·DOI

5

Radiosensitization and DNA repair inhibition by the combined use of novel inhibitors of DNA-dependent protein kinase and poly(ADP-ribose) polymerase-1.

PubMed·2003·331 cit.

6

Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiency

Molecular Cell·2021·315 cit.·DOI

7

Inferring and perturbing cell fate regulomes in human brain organoids

Nature·2022·309 cit.·DOI

8

VPS35 in Dopamine Neurons Is Required for Endosome-to-Golgi Retrieval of Lamp2a, a Receptor of Chaperone-Mediated Autophagy That Is Critical for -Synuclein Degradation and Prevention of Pathogenesis of Parkinson's Disease

Journal of Neuroscience·2015·270 cit.·DOI

9

Inhibition of Drp1 Ameliorates Synaptic Depression, Aβ Deposition, and Cognitive Impairment in an Alzheimer's Disease Model

Journal of Neuroscience·2017·257 cit.·DOI

10

Activation of the MAPK pathway is a common event in uveal melanomas although it rarely occurs through mutation of BRAF or RAS

British Journal of Cancer·2005·217 cit.·DOI

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