Cell Signaling Technology

UCP1 (D9D6X) Rabbit Monoclonal Antibody

#14670 · $161,351

61

Evidence Score · Well-Established

Used by 104 independent labs across 118 papers. How we score this →

100

Citing papers

4.5k

Combined citations

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Research areas

Alzheimer's disease research and treatments14 Adipose Tissue and Metabolism9 Neuroscience and Neuropharmacology Research8 Autophagy in Disease and Therapy8 Mitochondrial Function and Pathology8 Cholinesterase and Neurodegenerative Diseases6 Adipokines, Inflammation, and Metabolic Diseases6 Pancreatic function and diabetes5

Papers citing this product

1

β-Hydroxybutyrate inhibits inflammasome activation to attenuate Alzheimer’s disease pathology

Journal of Neuroinflammation·2020·243 cit.·DOI

2

A dysregulated bile acid-gut microbiota axis contributes to obesity susceptibility

EBioMedicine·2020·238 cit.·DOI

3

Notch signaling in chondrocytes modulates endochondral ossification and osteoarthritis development

Proceedings of the National Academy of Sciences·2013·201 cit.·DOI

4

Expression of oxytocin, oestrogen and progesterone receptors in uterine biopsy samples throughout the oestrous cycle and early pregnancy in cows

Reproduction·2001·200 cit.·DOI

5

Dynamic Regulation of CFTR Bicarbonate Permeability by [Cl−]i and Its Role in Pancreatic Bicarbonate Secretion

Gastroenterology·2010·193 cit.·DOI

6

Notch Is a Critical Component of the Mouse Somitogenesis Oscillator and Is Essential for the Formation of the Somites

PLoS Genetics·2009·124 cit.·DOI

7

IGF-I and IGFBP-2 Stimulate AMPK Activation and Autophagy, Which Are Required for Osteoblast Differentiation

Endocrinology·2015·120 cit.·DOI

8

Cyclophilin D Deficiency Rescues Axonal Mitochondrial Transport in Alzheimer’s Neurons

PLoS ONE·2013·118 cit.·DOI

9

Crucial role of calbindin-D28k in the pathogenesis of Alzheimer’s disease mouse model

Cell Death and Differentiation·2014·112 cit.·DOI

10

Phenylpiperidine‐type γ‐secretase modulators target the transmembrane domain 1 of presenilin 1

The EMBO Journal·2011·111 cit.·DOI

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