Cell Signaling Technology

AMPK alpha (23A3) Rabbit Monoclonal Antibody

#2603 · $157,348

64
Evidence Score · Well-Established
Used by 692 independent labs across 756 papers. How we score this →
100
Citing papers
21k
Combined citations
Supplier page Find this product

Research areas

Adipose Tissue and Metabolism10Autophagy in Disease and Therapy9Fibroblast Growth Factor Research7Pancreatic function and diabetes6Cytokine Signaling Pathways and Interactions6Metabolism, Diabetes, and Cancer6Nerve injury and regeneration6Immune cells in cancer5

Papers citing this product

1
FGF-21 as a novel metabolic regulator
Journal of Clinical Investigation·2005·2.1k cit.·DOI
2
Proteogenomic Characterization Reveals Therapeutic Vulnerabilities in Lung Adenocarcinoma
Cell·2020·811 cit.·DOI
3
Transcriptional control of autophagy–lysosome function drives pancreatic cancer metabolism
Nature·2015·788 cit.·DOI
4
Research Resource: Comprehensive Expression Atlas of the Fibroblast Growth Factor System in Adult Mouse
Molecular Endocrinology·2010·679 cit.·DOI
5
Fibroblast Growth Factor-21 Improves Pancreatic β-Cell Function and Survival by Activation of Extracellular Signal–Regulated Kinase 1/2 and Akt Signaling Pathways
Diabetes·2006·491 cit.·DOI
6
SirT1 Regulates Energy Metabolism and Response to Caloric Restriction in Mice
PLoS ONE·2008·440 cit.·DOI
7
RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury
Cell Death and Differentiation·2016·439 cit.·DOI
8
Cancer Mutations of the Tumor Suppressor SPOP Disrupt the Formation of Active, Phase-Separated Compartments
Molecular Cell·2018·411 cit.·DOI
9
Baicalin ameliorates neuroinflammation-induced depressive-like behavior through inhibition of toll-like receptor 4 expression via the PI3K/AKT/FoxO1 pathway
Journal of Neuroinflammation·2019·407 cit.·DOI
10
MicroRNA-33–dependent regulation of macrophage metabolism directs immune cell polarization in atherosclerosis
Journal of Clinical Investigation·2015·385 cit.·DOI

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