Cell Signaling Technology

Myc-Tag (9B11) Mouse Monoclonal Antibody (Alexa Fluor® 555 Conjugate)

#3756 · $465

100
Citing papers
17.2k
Combined citations
Supplier page Find this product

Research areas

MicroRNA in disease regulation22Circular RNAs in diseases14Cancer-related molecular mechanisms research10Ubiquitin and proteasome pathways8Wnt/β-catenin signaling in development and cancer6Cancer Cells and Metastasis6Cancer, Hypoxia, and Metabolism6Cell Adhesion Molecules Research5

Papers citing this product

1
MicroRNA profiling reveals distinct signatures in B cell chronic lymphocytic leukemias
Proceedings of the National Academy of Sciences·2004·1.3k cit.·DOI
2
miR-124 and miR-137 inhibit proliferation of glioblastoma multiforme cells and induce differentiation of brain tumor stem cells
BMC Medicine·2008·948 cit.·DOI
3
FGF stimulation of the Erk1/2 signalling cascade triggers transition of pluripotent embryonic stem cells from self-renewal to lineage commitment
Development·2007·805 cit.·DOI
4
Gain-of-function human <i>STAT1</i> mutations impair IL-17 immunity and underlie chronic mucocutaneous candidiasis
The Journal of Experimental Medicine·2011·797 cit.·DOI
5
Circular RNA circ-ITCH inhibits bladder cancer progression by sponging miR-17/miR-224 and regulating p21, PTEN expression
Molecular Cancer·2018·576 cit.·DOI
6
Leptin targets in the mouse brain
The Journal of Comparative Neurology·2009·481 cit.·DOI
7
Dual role of transcription factor FoxO1 in controlling hepatic insulin sensitivity and lipid metabolism
Journal of Clinical Investigation·2006·436 cit.·DOI
8
Epithelial to mesenchymal transition markers expressed in circulating tumour cells of early and metastatic breast cancer patients
Breast Cancer Research·2011·373 cit.·DOI
9
Metformin reduces liver glucose production by inhibition of fructose-1-6-bisphosphatase
Nature Medicine·2018·324 cit.·DOI
10
Smad2 and Smad3 Are Redundantly Essential for the TGF-β–Mediated Regulation of Regulatory T Plasticity and Th1 Development
The Journal of Immunology·2010·323 cit.·DOI

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