Cell Signaling Technology

SHP-2 (D50F2) Rabbit Monoclonal Antibody (BSA and Azide Free)

#31259 · $865

16

Citing papers

721

Combined citations

Supplier page Find this product

Research areas

RNA modifications and cancer3 Effects and risks of endocrine disrupting chemicals3 Tryptophan and brain disorders2 Adipose Tissue and Metabolism2 Mitochondrial Function and Pathology2 Autophagy in Disease and Therapy2 Epigenetics and DNA Methylation2 Cancer, Hypoxia, and Metabolism1

Papers citing this product

1

ATF4 suppresses hepatocarcinogenesis by inducing SLC7A11 (xCT) to block stress-related ferroptosis

Journal of Hepatology·2023·356 cit.·DOI

2

Targeting NEK2 attenuates glioblastoma growth and radioresistance by destabilizing histone methyltransferase EZH2

Journal of Clinical Investigation·2017·101 cit.·DOI

3

Single-cell transcriptomic dissection of the cellular and molecular events underlying the triclosan-induced liver fibrosis in mice

Military Medical Research·2023·51 cit.·DOI

4

Drosophila MFAP1 Is Required for Pre-mRNA Processing and G2/M Progression

Journal of Biological Chemistry·2008·43 cit.·DOI

5

A Spontaneous Mutation of the Rat Themis Gene Leads to Impaired Function of Regulatory T Cells Linked to Inflammatory Bowel Disease

PLoS Genetics·2012·39 cit.·DOI

6

Phosphodiesterase 3B (PDE3B) regulates NLRP3 inflammasome in adipose tissue

Scientific Reports·2016·36 cit.·DOI

7

Lactational delivery of Triclosan promotes non-alcoholic fatty liver disease in newborn mice

Nature Communications·2022·28 cit.·DOI

8

PolG Inhibits Gastric Cancer Glycolysis and Viability by Suppressing PKM2 Phosphorylation

Cancer Management and Research·2021·20 cit.·DOI

9

Natural variation in age-related dopamine neuron degeneration is glutathione dependent and linked to life span

Proceedings of the National Academy of Sciences·2024·13 cit.·DOI

10

Dachaihu decoction inhibits hypernutrition-induced liver metastasis from colorectal cancer by maintaining the gut vascular barrier

Cancer Pathogenesis and Therapy·2023·9 cit.·DOI

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