Cell Signaling Technology

Neurofilament-L (C28E10) Rabbit Monoclonal Antibody

#2837 · western blot · $152,351

66
Evidence Score · Well-Established
Used by 595 independent labs across 726 papers. How we score this →
100
Citing papers
13.7k
Combined citations
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Research areas

Autophagy in Disease and Therapy11Glycosylation and Glycoproteins Research7Microtubule and mitosis dynamics5Epigenetics and DNA Methylation5DNA Repair Mechanisms5Cell death mechanisms and regulation5Mesenchymal stem cell research5Neurogenesis and neuroplasticity mechanisms5

Papers citing this product

1
Identification of RNF213 as a Susceptibility Gene for Moyamoya Disease and Its Possible Role in Vascular Development
PLoS ONE·2011·711 cit.·DOI
2
Dietary tryptophan metabolite released by intratumoral Lactobacillus reuteri facilitates immune checkpoint inhibitor treatment
Cell·2023·635 cit.·DOI
3
Genetic insights into biological mechanisms governing human ovarian ageing
Nature·2021·442 cit.·DOI
4
SOX11 expression is highly specific for mantle cell lymphoma and identifies the cyclin D1-negative subtype
Haematologica·2009·401 cit.·DOI
5
A cancer vaccine-mediated postoperative immunotherapy for recurrent and metastatic tumors
Nature Communications·2018·394 cit.·DOI
6
Glycogen Synthase Kinase-3β Phosphorylates Bax and Promotes Its Mitochondrial Localization during Neuronal Apoptosis
Journal of Neuroscience·2004·361 cit.·DOI
7
DNA damaging agent-induced autophagy produces a cytoprotective adenosine triphosphate surge in malignant glioma cells
Cell Death and Differentiation·2006·356 cit.·DOI
8
Acetylation of MORC2 by NAT10 regulates cell-cycle checkpoint control and resistance to DNA-damaging chemotherapy and radiotherapy in breast cancer
Nucleic Acids Research·2020·228 cit.·DOI
9
Cocaine-Induced Intracellular Signaling and Gene Expression Are Oppositely Regulated by the Dopamine D1 and D3 Receptors
Journal of Neuroscience·2004·226 cit.·DOI
10
Astrocytic tight junctions control inflammatory CNS lesion pathogenesis
Journal of Clinical Investigation·2017·219 cit.·DOI

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