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Cell Signaling Technology
Phospho-Lyn (Tyr507) Antibody
#2731 · $148
100
Citing papers
27.6k
Combined citations
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Research areas
Autophagy in Disease and Therapy
11
DNA Repair Mechanisms
8
Immune Response and Inflammation
7
Cancer-related molecular mechanisms research
7
Cancer, Hypoxia, and Metabolism
6
MicroRNA in disease regulation
6
RNA modifications and cancer
6
Cell death mechanisms and regulation
5
Papers citing this product
1
Heat Shock Protein A12B Protects Vascular Endothelial Cells Against Sepsis-Induced Acute Lung Injury in Mice
Cellular Physiology and Biochemistry
·
2017
·
10.7k cit.
·
DOI
2
A New Mesenchymal Stem Cell (MSC) Paradigm: Polarization into a Pro-Inflammatory MSC1 or an Immunosuppressive MSC2 Phenotype
PLoS ONE
·
2010
·
1.2k cit.
·
DOI
3
Atezolizumab in combination with bevacizumab enhances antigen-specific T-cell migration in metastatic renal cell carcinoma
Nature Communications
·
2016
·
708 cit.
·
DOI
4
Metformin Enhances Autophagy and Normalizes Mitochondrial Function to Alleviate Aging-Associated Inflammation
Cell Metabolism
·
2020
·
597 cit.
·
DOI
5
Cancer Mutations of the Tumor Suppressor SPOP Disrupt the Formation of Active, Phase-Separated Compartments
Molecular Cell
·
2018
·
411 cit.
·
DOI
6
Lipopolysaccharide activation of the MEK-ERK1/2 pathway in human monocytic cells mediates tissue factor and tumor necrosis factor α expression by inducing Elk-1 phosphorylation and Egr-1 expression
Blood
·
2001
·
368 cit.
·
DOI
7
Critical Role for the NLRP3 Inflammasome during Acute Lung Injury
The Journal of Immunology
·
2014
·
349 cit.
·
DOI
8
The complex structure of GRL0617 and SARS-CoV-2 PLpro reveals a hot spot for antiviral drug discovery
Nature Communications
·
2021
·
340 cit.
·
DOI
9
Enhanced PIP3 signaling in POMC neurons causes KATP channel activation and leads to diet-sensitive obesity
Journal of Clinical Investigation
·
2006
·
322 cit.
·
DOI
10
KRAS mutation-induced upregulation of PD-L1 mediates immune escape in human lung adenocarcinoma
Cancer Immunology Immunotherapy
·
2017
·
292 cit.
·
DOI
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