Cell Signaling Technology

PI3 Kinase p110 gamma Antibody

#4252 · $317

100
Citing papers
12k
Combined citations
Supplier page Find this product

Research areas

NF-κB Signaling Pathways8Estrogen and related hormone effects7Immune Response and Inflammation7Effects and risks of endocrine disrupting chemicals5Heme Oxygenase-1 and Carbon Monoxide5Alzheimer's disease research and treatments5Genomics, phytochemicals, and oxidative stress5Glycosylation and Glycoproteins Research5

Papers citing this product

1
Suppression of canonical Wnt/ -catenin signaling by nuclear plakoglobin recapitulates phenotype of arrhythmogenic right ventricular cardiomyopathy
Journal of Clinical Investigation·2006·614 cit.·DOI
2
Functional Impairment of Microglia Coincides with Beta-Amyloid Deposition in Mice with Alzheimer-Like Pathology
PLoS ONE·2013·477 cit.·DOI
3
Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress
Science·2013·410 cit.·DOI
4
Activation of the Akt-NF-κB Pathway by Subtilase Cytotoxin through the ATF6 Branch of the Unfolded Protein Response
The Journal of Immunology·2009·283 cit.·DOI
5
Exosomal transfer of long non-coding RNA SBF2-AS1 enhances chemoresistance to temozolomide in glioblastoma
Journal of Experimental & Clinical Cancer Research·2019·266 cit.·DOI
6
Enhancing Astrocytic Lysosome Biogenesis Facilitates Aβ Clearance and Attenuates Amyloid Plaque Pathogenesis
Journal of Neuroscience·2014·265 cit.·DOI
7
Increased activation of p38 MAPK in COPD
European Respiratory Journal·2007·261 cit.·DOI
8
UCP2-induced fatty acid synthase promotes NLRP3 inflammasome activation during sepsis
Journal of Clinical Investigation·2015·239 cit.·DOI
9
Differential Estrogenic Actions of Endocrine-Disrupting Chemicals Bisphenol A, Bisphenol AF, and Zearalenone through Estrogen Receptor α and β <i>in Vitro</i>
Environmental Health Perspectives·2012·229 cit.·DOI
10
IFN-γ Promotes Complement Expression and Attenuates Amyloid Plaque Deposition in Amyloid β Precursor Protein Transgenic Mice
The Journal of Immunology·2010·227 cit.·DOI

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