Cell Signaling Technology

SMG-1 (Q25) Antibody

#4993 · $317

100
Citing papers
8k
Combined citations
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Research areas

Cancer-related Molecular Pathways14Neurogenesis and neuroplasticity mechanisms10Epigenetics and DNA Methylation8DNA Repair Mechanisms7Endoplasmic Reticulum Stress and Disease6Ubiquitin and proteasome pathways6Neuroscience and Neuropharmacology Research6Cell Adhesion Molecules Research5

Papers citing this product

1
Lactate increases stemness of CD8 + T cells to augment anti-tumor immunity
Nature Communications·2022·384 cit.·DOI
2
The combined status of ATM and p53 link tumor development with therapeutic response
Genes & Development·2009·301 cit.·DOI
3
Activation of ATM/Chk1 by curcumin causes cell cycle arrest and apoptosis in human pancreatic cancer cells
British Journal of Cancer·2009·216 cit.·DOI
4
Lrp4 Modulates Extracellular Integration of Cell Signaling Pathways in Development
PLoS ONE·2008·201 cit.·DOI
5
Loss of Raf Kinase Inhibitor Protein Promotes Cell Proliferation and Migration of Human Hepatoma Cells
Gastroenterology·2006·189 cit.·DOI
6
Human umbilical cord-derived mesenchymal stem cells elicit macrophages into an anti-inflammatory phenotype to alleviate insulin resistance in type 2 diabetic rats
Stem Cells·2015·188 cit.·DOI
7
Role of Pro-oncogenic Protein Disulfide Isomerase (PDI) Family Member Anterior Gradient 2 (AGR2) in the Control of Endoplasmic Reticulum Homeostasis
Journal of Biological Chemistry·2011·172 cit.·DOI
8
Tumor-draining lymph node is important for a robust abscopal effect stimulated by radiotherapy
Journal for ImmunoTherapy of Cancer·2020·170 cit.·DOI
9
The Proteasome Inhibitor PS-341 (Bortezomib) Up-Regulates DR5 Expression Leading to Induction of Apoptosis and Enhancement of TRAIL-Induced Apoptosis Despite Up-Regulation of c-FLIP and Survivin Expression in Human NSCLC Cells
Cancer Research·2007·164 cit.·DOI
10
Cyclin A2/E1 activation defines a hepatocellular carcinoma subclass with a rearrangement signature of replication stress
Nature Communications·2018·150 cit.·DOI

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