Cell Signaling Technology

PYURF (F9F4X) Rabbit Monoclonal Antibody

#16847 · western blot · $161,376

76

Citing papers

4.5k

Combined citations

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Research areas

Parkinson's Disease Mechanisms and Treatments39 Cellular transport and secretion14 Lysosomal Storage Disorders Research8 Plant Gene Expression Analysis8 Fungal and yeast genetics research7 Neurological diseases and metabolism7 Retinal Development and Disorders6 Autophagy in Disease and Therapy4

Papers citing this product

1

LRRK2 Inhibition Attenuates Microglial Inflammatory Responses

Journal of Neuroscience·2012·473 cit.·DOI

2

14-3-3 binding to LRRK2 is disrupted by multiple Parkinson's disease-associated mutations and regulates cytoplasmic localization

Biochemical Journal·2010·402 cit.·DOI

3

Enhanced Striatal Dopamine Transmission and Motor Performance with LRRK2 Overexpression in Mice Is Eliminated by Familial Parkinson's Disease Mutation G2019S

Journal of Neuroscience·2010·344 cit.·DOI

4

Leucine-rich repeat kinase 2 regulates autophagy through a calcium-dependent pathway involving NAADP

Human Molecular Genetics·2011·316 cit.·DOI

5

LRRK2 Controls Synaptic Vesicle Storage and Mobilization within the Recycling Pool

Journal of Neuroscience·2011·268 cit.·DOI

6

The IkappaB Kinase Family Phosphorylates the Parkinson’s Disease Kinase LRRK2 at Ser935 and Ser910 during Toll-Like Receptor Signaling

PLoS ONE·2012·215 cit.·DOI

7

Phos-tag analysis of Rab10 phosphorylation by LRRK2: a powerful assay for assessing kinase function and inhibitors

Biochemical Journal·2016·181 cit.·DOI

8

Altered Development of Synapse Structure and Function in Striatum Caused by Parkinson's Disease-Linked LRRK2-G2019S Mutation

Journal of Neuroscience·2016·149 cit.·DOI

9

Association of Interleukin-6 Signalling with the Muscle Stem Cell Response Following Muscle-Lengthening Contractions in Humans

PLoS ONE·2009·146 cit.·DOI

10

Mutant LRRK2 Toxicity in Neurons Depends on LRRK2 Levels and Synuclein But Not Kinase Activity or Inclusion Bodies

Journal of Neuroscience·2014·140 cit.·DOI

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