Cell Signaling Technology

Phospho-MKK3 (Ser189)/MKK6 (Ser207) Antibody

#9231 · western blot · $362

64
Evidence Score · Well-Established
Used by 280 independent labs across 301 papers. How we score this →
100
Citing papers
11k
Combined citations
Supplier page Find this product

Research areas

Autophagy in Disease and Therapy49Endoplasmic Reticulum Stress and Disease47Pancreatic function and diabetes8Sirtuins and Resveratrol in Medicine5Cancer Mechanisms and Therapy4RNA regulation and disease4Phagocytosis and Immune Regulation4Cancer, Hypoxia, and Metabolism3

Papers citing this product

1
The eIF2α/ATF4 pathway is essential for stress-induced autophagy gene expression
Nucleic Acids Research·2013·1.1k cit.·DOI
2
The unfolded protein response protects human tumor cells during hypoxia through regulation of the autophagy genes MAP1LC3B and ATG5
Journal of Clinical Investigation·2009·799 cit.·DOI
3
Autophagy in Idiopathic Pulmonary Fibrosis
PLoS ONE·2012·378 cit.·DOI
4
KIM-1–mediated phagocytosis reduces acute injury to the kidney
Journal of Clinical Investigation·2015·356 cit.·DOI
5
Mammalian target of rapamycin and glycogen synthase kinase 3 differentially regulate lipopolysaccharide-induced interleukin-12 production in dendritic cells
Blood·2008·262 cit.·DOI
6
DAP-kinase is a mediator of endoplasmic reticulum stress-induced caspase activation and autophagic cell death
Cell Death and Differentiation·2008·251 cit.·DOI
7
HSP72 Protects Cells from ER Stress-induced Apoptosis via Enhancement of IRE1α-XBP1 Signaling through a Physical Interaction
PLoS Biology·2010·238 cit.·DOI
8
Transplantation of cardiac progenitor cells ameliorates cardiac dysfunction after myocardial infarction in mice
Journal of Clinical Investigation·2009·236 cit.·DOI
9
Neutrophil extracellular trap induced by HMGB1 exacerbates damages in the ischemic brain
Acta Neuropathologica Communications·2019·223 cit.·DOI
10
Golgi apparatus-targeted aggregation-induced emission luminogens for effective cancer photodynamic therapy
Nature Communications·2022·209 cit.·DOI

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