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Cell Signaling Technology
Phospho-PAR-4 (Thr163) Antibody
#2329 · $362
100
Citing papers
18.6k
Combined citations
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Research areas
MicroRNA in disease regulation
8
Cell death mechanisms and regulation
7
PI3K/AKT/mTOR signaling in cancer
6
Cancer-related molecular mechanisms research
6
RNA modifications and cancer
6
Cancer Immunotherapy and Biomarkers
5
Epigenetics and DNA Methylation
5
Autophagy in Disease and Therapy
5
Papers citing this product
1
Recurrent somatic alterations of FGFR1 and NTRK2 in pilocytic astrocytoma
Nature Genetics
·
2013
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835 cit.
·
DOI
2
Microbiota-derived short-chain fatty acids promote Th1 cell IL-10 production to maintain intestinal homeostasis
Nature Communications
·
2018
·
656 cit.
·
DOI
3
Self-Organizing 3D Human Neural Tissue Derived from Induced Pluripotent Stem Cells Recapitulate Alzheimer’s Disease Phenotypes
PLoS ONE
·
2016
·
573 cit.
·
DOI
4
PKM2 regulates the Warburg effect and promotes HMGB1 release in sepsis
Nature Communications
·
2014
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511 cit.
·
DOI
5
BIM Mediates EGFR Tyrosine Kinase Inhibitor-Induced Apoptosis in Lung Cancers with Oncogenic EGFR Mutations
PLoS Medicine
·
2007
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490 cit.
·
DOI
6
Single-Cell Transcriptomics Uncovers Zonation of Function in the Mesenchyme during Liver Fibrosis
Cell Reports
·
2019
·
420 cit.
·
DOI
7
Adipocyte lipolysis links obesity to breast cancer growth: adipocyte-derived fatty acids drive breast cancer cell proliferation and migration
Cancer & Metabolism
·
2017
·
413 cit.
·
DOI
8
MYC pathway activation in triple-negative breast cancer is synthetic lethal with CDK inhibition
The Journal of Experimental Medicine
·
2012
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395 cit.
·
DOI
9
Chronic oral application of a periodontal pathogen results in brain inflammation, neurodegeneration and amyloid beta production in wild type mice
PLoS ONE
·
2018
·
360 cit.
·
DOI
10
Eribulin mesilate suppresses experimental metastasis of breast cancer cells by reversing phenotype from epithelial–mesenchymal transition (EMT) to mesenchymal–epithelial transition (MET) states
British Journal of Cancer
·
2014
·
355 cit.
·
DOI
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