Cell Signaling Technology

MMP-3 (D7F5B) Rabbit Monoclonal Antibody

#14351 · $161

100
Citing papers
3k
Combined citations
Supplier page Find this product

Research areas

Ferroptosis and cancer prognosis15RNA modifications and cancer14Epigenetics and DNA Methylation11Hippo pathway signaling and YAP/TAZ9Cancer-related molecular mechanisms research7MicroRNA in disease regulation7Protein Kinase Regulation and GTPase Signaling6Protease and Inhibitor Mechanisms6

Papers citing this product

1
ATF4 suppresses hepatocarcinogenesis by inducing SLC7A11 (xCT) to block stress-related ferroptosis
Journal of Hepatology·2023·356 cit.·DOI
2
Cancer‑associated fibroblast‑derived exosomal miR‑382‑5p promotes the migration and invasion of oral squamous cell carcinoma
Oncology Reports·2019·168 cit.·DOI
3
Imatinib inhibits VEGF-independent angiogenesis by targeting neuropilin 1–dependent ABL1 activation in endothelial cells
The Journal of Experimental Medicine·2014·136 cit.·DOI
4
CircRNA-CIDN mitigated compression loading-induced damage in human nucleus pulposus cells via miR-34a-5p/SIRT1 axis
EBioMedicine·2020·124 cit.·DOI
5
Annexin A2 Promotes Glioma Cell Invasion and Tumor Progression
Journal of Neuroscience·2011·117 cit.·DOI
6
Restoration of Autophagic Flux Rescues Oxidative Damage and Mitochondrial Dysfunction to Protect against Intervertebral Disc Degeneration
Oxidative Medicine and Cellular Longevity·2019·116 cit.·DOI
7
Salmonella Modulation of Host Cell Gene Expression Promotes Its Intracellular Growth
PLoS Pathogens·2013·79 cit.·DOI
8
RUNX2 recruits the NuRD(MTA1)/CRL4B complex to promote breast cancer progression and bone metastasis
Cell Death and Differentiation·2022·73 cit.·DOI
9
Cathepsin C Interacts with TNF-α/p38 MAPK Signaling Pathway to Promote Proliferation and Metastasis in Hepatocellular Carcinoma
Cancer Research and Treatment·2019·72 cit.·DOI
10
Amyloid precursor protein promotes the migration and invasion of breast cancer cells by regulating the MAPK signaling pathway
International Journal of Molecular Medicine·2019·62 cit.·DOI

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